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A gene variant increases the risk of a type of hair loss by affecting hair protein production.

The gene Tfap2b is essential for creating a type of stem cell in zebrafish that can become different pigment cells.

Cells lacking the Bax protein can outcompete others, leading to better tissue repair and hair growth.

A mutation in the EDNRA gene causes Oro-Oto-Cardiac syndrome, affecting face and heart development.

A special gene region controls the re-emergence of a primitive wool type in Merino sheep, improving their wool yield and adaptability.

Atopic dermatitis shows a link between skin layers in inflammation, detectable with detailed gene analysis.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

The extracellular matrix affects where tumors can start in the body.

Blocking testosterone speeds up wound healing in males.

Blocking testosterone speeds up wound healing in males.

Progerin affects cell shape but not hair or skin in mice.

Mitochondrial dysfunction contributes to skin aging and hair loss.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Diabetic neuropathy in mice is linked to poor mitochondria function and lower brain hormone production.

Enzymes involved in Vitamin A metabolism affect hair growth and type in mice.

Turning white fat into brown-like fat could help fight obesity and type 2 diabetes.

Citrullinated proteins from Porphyromonas gingivalis may contribute to rheumatoid arthritis.

Keratin 17 is important for skin's response to radiation, affecting many genes and cell division.

Removing certain brain receptors in mice worsens seizure severity and response to treatment during hormone withdrawal.

Blocking TRPV3 may help treat itch in dry skin conditions.

Androgen self-administration might be controlled by membrane receptors, not nuclear ones.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

Certain mice without specific receptors or mast cells don't lose hair from stress.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Bcl-2 overexpression protects against UVB damage but worsens hair loss from chemotherapy.

Mice with more Flightless I protein grew back their claws better after amputation.

Not having enough cystatin M/E protein causes less hair growth and dry skin.

Kir6.1 mutations in Cantú syndrome increase channel sensitivity and hyperpolarization, while SUR2B mutations do not.