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Research 901–930 of 1000+
- Directed elimination of senescent cells by inhibition of BCL-W and BCL-XL
- c-Myc activation in transgenic mouse epidermis results in mobilization of stem cells and differentiation of their progeny
- Regulation of somatic cell reprogramming through inducible mir-302 expression
- Targeted Inactivation of Gh/Tissue Transglutaminase II
- Intradermal adipocytes mediate fibroblast recruitment during skin wound healing
- Keratin 15 Expression in Stratified Epithelia: Downregulation in Activated Keratinocytes
- Mice lacking desmocollin 1 show epidermal fragility accompanied by barrier defects and abnormal differentiation
- Very-long-chain Acyl-CoA Synthetases
- Flavonoids as Potential Wound-Healing Molecules: Emphasis on Pathways Perspective
- GLI2 Is Expressed in Normal Human Epidermis and BCC and Induces GLI1 Expression by Binding to its Promoter
- p63 regulates <i>Satb1</i> to control tissue-specific chromatin remodeling during development of the epidermis
- Wnt signaling induces epithelial differentiation during cutaneous wound healing
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Human adipose-derived mesenchymal stem cells-derived exosomes encapsulated in pluronic F127 hydrogel promote wound healing and regeneration
- Characterization of Hair Follicle Antigens Targeted by the Anti-Hair Follicle Immune Response
- Premature Senescence of Balding Dermal Papilla Cells In Vitro Is Associated with p16INK4a Expression
- Human bone marrow mesenchymal stem cell-derived exosomes stimulate cutaneous wound healing mediates through TGF-β/Smad signaling pathway
- Shh maintains dermal papilla identity and hair morphogenesis via a Noggin–Shh regulatory loop
- Epithelial Integrity Is Maintained by a Matriptase-Dependent Proteolytic Pathway
- Hyaluronate Fragments Reverse Skin Atrophy by a CD44-Dependent Mechanism
- The Vitamin D Receptor Is a Wnt Effector that Controls Hair Follicle Differentiation and Specifies Tumor Type in Adult Epidermis
- TRP Channels in Skin Biology and Pathophysiology
- MicroRNA-302 Increases Reprogramming Efficiency via Repression of NR2F2
- Mutations in Three Genes Encoding Proteins Involved in Hair Shaft Formation Cause Uncombable Hair Syndrome
- The Tissue-dependent Keratin 19 Gene Transcription Is Regulated by GKLF/KLF4 and Sp1
- Macrophages induce AKT/β-catenin-dependent Lgr5+ stem cell activation and hair follicle regeneration through TNF
- Cytochrome P450: A Target for Drug Development for Skin Diseases
- Hyperandrogenism and insulin resistance modulate gravid uterine and placental ferroptosis in PCOS-like rats
- Conditional epidermal expression of TGFβ1 blocks neonatal lethality but causes a reversible hyperplasia and alopecia
- A Subset of TREM2+ Dermal Macrophages Secretes Oncostatin M to Maintain Hair Follicle Stem Cell Quiescence and Inhibit Hair Growth