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Intravenous and oral calcium effectively treated vitamin D dependent rickets type II, improving growth and bone health.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

Madarosis is the loss of eyelashes and eyebrows due to various health issues and requires thorough examination to diagnose and treat the underlying cause.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

A boy with severe Vitamin D-resistant rickets did not respond to treatment and lacked a common symptom, suggesting a need for alternative treatments.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

Hair loss can be a key sign of a rare type of rickets when vitamin D treatment doesn't work and advanced tests aren't available.

A genetic mutation caused severe rickets and alopecia in an Indian patient, but high-dose calcium and phosphate treatment improved their condition.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

A 4-year-old boy with a rare type of rickets and hair loss improved in bone health but not hair growth after vitamin D and calcium treatment.

A boy's hair loss and bone issues improved with specific vitamin D treatment and calcium.

Two siblings stayed rickets-free for 14 years after stopping treatment.

Child with rickets improved with a specific vitamin D therapy, but alopecia did not change.

Vitamin D receptor is crucial for bone health and mineral metabolism.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Vitamin D is crucial for bone health and preventing serious diseases.

Researchers created a mouse model of a type of rickets that does not cause hair loss.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Genetic testing is crucial for diagnosing congenital atrichia, a rare condition causing irreversible hair loss.

A girl with rickets and hair loss was found to have a rare condition where her body didn't respond to vitamin D treatment.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.