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Research 91–120 of 1000+
- Rickets in association with skin diseases and conditions: A review with emphasis on screening and prevention
- In vivo function of VDR in gene expression-VDR knock-out mice
- Editorial: Vitamin D 1α-Hydroxylase Knockout Mice as a Hereditary Rickets Animal Model
- 4 Vitamin D resistance
- Receptors in trichology: Hot spots for hair regeneration
- Atrichia congenita with papular lesions: A rare cause of pediatric alopecia
- Tooth Development Associated with Mutations in Hereditary Vitamin D–Resistant Rickets
- A human vitamin D receptor mutation causes rickets and impaired Th1/Th17 responses
- Ligand‐independent Regulation of the hairless Promoter by Vitamin D Receptor<sup>†</sup>
- Mutations in the vitamin D receptor gene in four patients with hereditary 1,25-dihydroxyvitamin D-resistant rickets
- Commonly associated disorders with complete scalp alopecia in early childhood: A review
- Congenital atrichia with papular lesions
- Mechanism of Vitamin D Receptor Action
- Generation of Genetically Modified Rats Using CRISPR/Cas9 Genome-Editing System to Reveal Novel Vitamin D Actions
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Hereditary Vitamin D Resistant Rickets: Clinical, Laboratory, and Genetic Characteristics of 2 Iranian Siblings
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia in four Egyptian families: report of three novel mutations in the vitamin D receptor gene
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Role of the vitamin D receptor in hair follicle biology
- Two siblings with a novel nonsense mutation, p.R50X, in the vitamin D receptor gene
- The role of vitamin D receptor mutations in the development of alopecia
- Rickets with alopecia: An inborn error of vitaminD metabolism
- 1,25-dihydroxyvitamin D resistance, rickets, and alopecia
- Prevalence and Etiology of Central Centrifugal Cicatricial Alopecia
- Extraskeletal actions of vitamin D
- Vitamin D Receptor Gene Polymorphism In Chronic Telogen Effluvium; A Case-Control Study
- Vitamin D increases expression of cathelicidin in cultured sebocytes
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Recomendations on non-pharmacological interventions in women with PCOS to reduce body weight and improve metabolic disorders [Zalecenia dotyczące postępowania niefarmakologicznego u kobiet z PCOS celem zmniejszenia masy ciała i poprawy zaburzeń metabolicznych]