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The review found no clear link between vitamin D receptor gene variations and polycystic ovary syndrome.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Four patients with a type of rickets and hair loss had different mutations in their vitamin D receptor gene, causing it to not work properly.

Low vitamin D receptor levels found in hair loss patients; topical vitamin D treatment suggested.

Vitamin D boosts a specific gene activity in kidney cells that could improve heart and kidney function.

Vitamin D deficiency is common in women with PCOS and linked to some metabolic problems, but not the main cause of their metabolic issues.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

People in Jeddah know about Vitamin D and its sources but are unclear on how much they need daily, and while many women take supplements, attitudes towards increasing Vitamin D levels vary.

Adequate vitamin D might lower, and high hair chromium might increase DNA damage in obese women.

A child with rickets and hair loss might have a rare type of rickets that doesn't improve much with usual vitamin D and calcium treatment.

A 3-year-old with vitamin D-resistant rickets and severe hair loss died despite treatment, highlighting the need for improved management of the condition.

A girl's hair-pulling condition improved greatly after she started vitamin D treatment.

Patients with Discoid Lupus Erythematosus have lower vitamin D and antioxidant levels and higher oxidative stress.

A child with a rare type of rickets showed some improvement with high doses of vitamin D, but such conditions often respond poorly to treatment.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Vitamin D levels are not correlated with chronic hair loss in Telogen Effluvium patients.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Vitamin D is important for skin health and can help treat psoriasis, atopic dermatitis, and vitiligo.

The study found that certain mutations in the vitamin D receptor can cause rickets and potentially affect hair growth.

A boy's hair loss and bone issues improved with specific vitamin D treatment and calcium.

Children with alopecia areata often have low vitamin D, especially if they have darker skin, it's not summer, or they're not White.

Vitamin D may help protect against nonmelanoma skin cancer.

Vitamin D is important for bones, hair, blood pressure, and breast development.

Two rare genetic diseases cause severe rickets in children due to defects in vitamin D metabolism.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Hairless protein can block vitamin D activation in skin cells.