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Research 601–630 of 1000+
- Regenerative medicine strategies for hair growth and regeneration: A narrative review of literature
- Vitamin D and Human Health: Lessons from Vitamin D Receptor Null Mice
- The Nuclear Vitamin D Receptor: Biological and Molecular Regulatory Properties Revealed
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice<sup>1</sup>
- Vitamin D Receptor: New Assignments for an Already Busy Receptor
- Vitamin D receptor: molecular signaling and actions of nutritional ligands in disease prevention
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- Lack of the Vitamin D Receptor is Associated with Reduced Epidermal Differentiation and Hair Follicle Growth
- Rickets with alopecia: An inborn error of vitaminD metabolism
- Behavioural characterization of Vitamin D receptor knockout mice
- Vitamin D and the skin: Physiology and pathophysiology
- Genetic Disorders and Defects in Vitamin D Action
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Ligand-Independent Actions of the Vitamin D Receptor Maintain Hair Follicle Homeostasis
- Vitamin D receptor is essential for normal keratinocyte stem cell function
- Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene
- Vitamin D Receptor: Key Roles in Bone Mineral Pathophysiology, Molecular Mechanism of Action, and Novel Nutritional Ligands
- Metabolic and cellular analysis of alopecia in vitamin D receptor knockout mice
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice
- Targeting Expression of the Human Vitamin D Receptor to the Keratinocytes of Vitamin D Receptor Null Mice Prevents Alopecia
- Evaluation of Keratinocyte Proliferation and Differentiation in Vitamin D Receptor Knockout Mice*
- Mechanism of Vitamin D Receptor Action
- The role of vitamin D receptor mutations in the development of alopecia
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Vitamin D deficiency in alopecia areata
- Cloning of a Functional Vitamin D Receptor from the Lamprey (Petromyzon marinus), an Ancient Vertebrate Lacking a Calcified Skeleton and Teeth
- Overexpression of Hedgehog Signaling Is Associated with Epidermal Tumor Formation in Vitamin D Receptor–Null Mice
- Vitamin D and aging
- Vitamin D and calcium regulation of epidermal wound healing
- An Unusual Form of Vitamin D-Dependent Rickets in a Child: Alopecia and Marked End-Organ Hyposensitivity to Biologically Active Vitamin D