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As skin cells mature, vitamin D receptor levels decrease while retinoid X receptor α levels increase.

Vitamin D3 may protect rat hair follicles from radiation damage.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor helps control hair growth genes in skin cells.

Combining skin tissue pathology with genetics has greatly improved the diagnosis and understanding of certain skin diseases.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Vitamin D receptor helps protect skin cells from UV damage and supports their growth.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

Tunisian children with hereditary vitamin D-resistant rickets showed improvement with calcium treatment, and new genetic mutations were identified.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Vitamin D and its receptor are important for hair and scalp health and may help treat hair loss.

Vitamin D helps skin stem cells heal wounds by working with a key skin protein.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

Vitamin D receptor is crucial for skin wound healing.

1,25-(OH)2D3 helps hair grow by blocking certain harmful signals.

The research showed that Vitamin D and its receptor are important for healthy bones and normal hair and skin in rats.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

KLF4 is important for keeping hair follicle stem cells inactive.

Vinblastine and its metabolites may cause nausea and hair loss by binding to specific receptors and could lead to better chemotherapy drugs with fewer side effects.

The vitamin D receptor helps maintain hair and bone health even without binding vitamin D.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

Vitamin D3 and its receptor are important for hair growth, and understanding receptors could help treat hair graying and skin cancer.

Vitamin D is made by the skin, helps control various body functions, and affects skin health and immunity.

The skin produces and uses vitamin D for bone health, cell growth, and immune function.