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Research 151–180 of 485
- Lack of the Vitamin D Receptor is Associated with Reduced Epidermal Differentiation and Hair Follicle Growth
- Behavioural characterization of Vitamin D receptor knockout mice
- Vitamin D and the skin: Physiology and pathophysiology
- Genetic Disorders and Defects in Vitamin D Action
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Ligand-Independent Actions of the Vitamin D Receptor Maintain Hair Follicle Homeostasis
- Vitamin D receptor is essential for normal keratinocyte stem cell function
- Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene
- Vitamin D Receptor: Key Roles in Bone Mineral Pathophysiology, Molecular Mechanism of Action, and Novel Nutritional Ligands
- Metabolic and cellular analysis of alopecia in vitamin D receptor knockout mice
- The Vitamin D Receptor Is a Wnt Effector that Controls Hair Follicle Differentiation and Specifies Tumor Type in Adult Epidermis
- Extraskeletal actions of vitamin D
- Normalization of Mineral Ion Homeostasis by Dietary Means Prevents Hyperparathyroidism, Rickets, and Osteomalacia, But Not Alopecia in Vitamin D Receptor-Ablated Mice
- Targeting Expression of the Human Vitamin D Receptor to the Keratinocytes of Vitamin D Receptor Null Mice Prevents Alopecia
- Evaluation of Keratinocyte Proliferation and Differentiation in Vitamin D Receptor Knockout Mice*
- Mechanism of Vitamin D Receptor Action
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Cloning of a Functional Vitamin D Receptor from the Lamprey (Petromyzon marinus), an Ancient Vertebrate Lacking a Calcified Skeleton and Teeth
- Overexpression of Hedgehog Signaling Is Associated with Epidermal Tumor Formation in Vitamin D Receptor–Null Mice
- Hair follicle expression of 1,25-dihydroxyvitamin D3 receptors during the murine hair cycle
- Vitamin D and calcium regulation of epidermal wound healing
- Development and progression of alopecia in the vitamin D receptor null mouse
- Hairless Suppresses Vitamin D Receptor Transactivation in Human Keratinocytes
- Hairless and Wnt Signaling: Allies in Epithelial Stem Cell Differentiation
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- Quantification of the Vitamin D Receptor−Coregulator Interaction
- Targeted Expression of Human Vitamin D Receptor in the Skin Promotes the Initiation of the Postnatal Hair Follicle Cycle and Rescues the Alopecia in Vitamin D Receptor Null Mice
- Vitamin D‐dependent rickets type I and type II
- The Vitamin D Receptor Is Required for Activation of cWnt and Hedgehog Signaling in Keratinocytes
- Ligand-Dependent Actions of the Vitamin D Receptor Are Required for Activation of TGF-β Signaling during the Inflammatory Response to Cutaneous Injury