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Research 30 of 496
- Analysis of hairless corepressor mutants to characterize molecular cooperation with the vitamin D receptor in promoting the mammalian hair cycle
- Analyse de la régulation du gène Hairless et identification des voies de signalisation affectées chez les mutants «bald Mill Hill».
- Hairless Modulates Ligand-Dependent Activation of the Vitamin D Receptor-Retinoid X Receptor Heterodimer
- Interactions of the Vitamin D Receptor with the Corepressor Hairless
- [Genetic dissection of retinoic acid function in epidermis physiology].
- Ligand-Independent Actions of the Vitamin D Receptor Maintain Hair Follicle Homeostasis
- The abnormal, mis-localizated HR bmh protein associates with members of the protein processing machinery in the cytoplasm
- Deletion of Deoxyribonucleic Acid Binding Domain of the Vitamin D Receptor Abrogates Genomic and Nongenomic Functions of Vitamin D
- A Humanized Mouse Model of Hereditary 1,25-Dihydroxyvitamin D–Resistant Rickets Without Alopecia
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Does Vitamin D play a role on Msx1 homeoprotein expression involving an endogenous antisense mRNA?*1
- Ligand-Independent Vitamin D Receptor Actions Essential for Keratinocyte Homeostasis in the Skin
- Hairless and Wnt Signaling: Allies in Epithelial Stem Cell Differentiation
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Hairless is a nuclear receptor corepressor essential for skin function
- The “Bald Mill Hill” Mutation in the Mouse Is Associated with an Abnormal, Mislocalized HR bmh Protein
- Ligand-Independent Vitamin D Receptor Actions Essential for Keratinocyte Homeostasis in the Skin
- Hereditary 1,25-Dihydroxyvitamin D Resistant Rickets due to a Mutation Causing Multiple Defects in Vitamin D Receptor Function
- Role of the vitamin D receptor in hair follicle biology
- Hypomorphic mutation in the hairless gene accelerates pruritic atopic skin caused by feeding a special diet to mice
- Disruption of the hedgehog signaling pathway contributes to the hair follicle cycling deficiency in Vdr knockout mice
- Regulation of VDR by ΔNp63α is associated with inhibition of cell invasion
- Mutant laboratory mice with abnormalities in hair follicle morphogenesis, cycling, and/or structure: An update
- Functional analyses of a novel missense and other mutations of the vitamin D receptor in association with alopecia
- In vivo function of VDR in gene expression-VDR knock-out mice
- Ligand‐independent Regulation of the hairless Promoter by Vitamin D Receptor<sup>†</sup>
- Generation of Genetically Modified Rats Using CRISPR/Cas9 Genome-Editing System to Reveal Novel Vitamin D Actions
- Functional analysis of vitamin D receptor (VDR) using adenovirus vector
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- Lymphoid Enhancer-binding Factor-1 (LEF1) Interacts with the DNA-binding Domain of the Vitamin D Receptor