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Research 31–60 of 35
- The Role of Nutrition in Immune-Mediated, Inflammatory Skin Disease: A Narrative Review
- Treatment of alopecia totalis/universalis/focalis with vitamin D and analogs: Three case reports and a literature review
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- The impact of VDR expression and regulation in vivo
- Diffuser Haarausfall und klinische Endokrinologie: Neue Erkenntnisse
- Vitamin D dependent rickets type 2: a case-based review of patient with alopecia and rickets
- Vitamin D-dependent rickets type II: extreme end organ resistance to 1,25-dihydroxy vitamin D3 in a patient without alopecia
- Vitamin D-dependent Rickets Type II
- A RARE CASE OF VITAMIN D DEPENDENT RICKETS TYPE-2A PRESENTED AS ALOPECIA TOTALIS
- Rickets with alopecia-remission following a course of 1-?-hydroxy vitamin D3 therapy
- Phenotypic Heterogeneity in 5 Japanese Patients with an Identical Point Mutation in the Vitamin D Receptor Gene
- Vitamin D‐dependent rickets type I and type II
- PO-0071 Role Of Growth Hormone Therapy In Clinical, Laboratory, Radiological Improvement Of Children With Vitamin-d Dependent Rickets Type-2
- Vitamin D Dependent Rickets Type II with Alopecia
- Genetic Disorders and Defects in Vitamin D Action
- Vitamin D-Dependent Rickets Type II with Alopecia: A Rare Case Report.
- Dental manifestations in Vitamin D dependent rickets Type II with Alopecia: a short pictorial case report
- An unusual presentation of vitamin D dependent rickets type 2 with low 25 (OH) D3 levels and alopecia: a case report of two siblings
- Type II Vitamin D Dependent Rickets: A Case Report
- Alopecia with Vitamin D-Dependent Rickets Type 2 A: A Case Report
- A rare case of Vitamin D dependent rickets type II: a case report
- In vivo function of VDR in gene expression-VDR knock-out mice
- Ligand‐independent Regulation of the hairless Promoter by Vitamin D Receptor<sup>†</sup>
- Presence of a Deletion Mutation (c.716delA) in the Ligand Binding Domain of the Vitamin D Receptor in an Indian Patient with Vitamin D-Dependent Rickets Type II
- ラット精巣におけるビタミンD 代謝関連遺伝子の発現解析
- Generation of Genetically Modified Rats Using CRISPR/Cas9 Genome-Editing System to Reveal Novel Vitamin D Actions
- Hereditary 1,25‐Dihydroxyvitamin D‐Resistant Rickets in a Pomeranian Dog Caused by a Novel Mutation in the Vitamin D Receptor Gene
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- A Humanized Mouse Model of Hereditary 1,25-Dihydroxyvitamin D–Resistant Rickets Without Alopecia
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3