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Research 31–60 of 1000+
- Polycystic ovarian syndrome
- Gonads
- The human keratins: biology and pathology
- Steroid Sulfatase: Molecular Biology, Regulation, and Inhibition
- Genetic, hormonal and metabolic aspects of PCOS: an update
- Roles for Msx and Dlx homeoproteins in vertebrate development
- Hormone replacement therapy and the skin
- Pharmacological evidences for the extracts and secondary metabolites from plants of the genus Hibiscus
- Tissue Engineering and Regenerative Medicine: Achievements, Future, and Sustainability in Asia
- A Novel Point Mutation in the Ligand-Binding Domain (LBD) of the Human Glucocorticoid Receptor (hGR) Causing Generalized Glucocorticoid Resistance: The Importance of the C Terminus of hGR LBD in Conferring Transactivational Activity
- Chapter 6 Fetal Skin Wound Healing
- Hair growth and rejuvenation: An overview
- Androgen receptors and their biology
- Nutrition Strategy and Life Style in Polycystic Ovary Syndrome—Narrative Review
- Androgen receptor gene CAG repeat polymorphism in women with polycystic ovary syndrome
- Novel cosmeceuticals from plants—An industry guided review
- Anabolic-androgenic steroids and cardiovascular risk
- Natural Molecules in the Management of Polycystic Ovary Syndrome (PCOS): An Analytical Review
- New approaches to the treatment of osteoporosis
- Androgen antagonists in androgen target tissues
- Fibroproliferative genes are preferentially expressed in central centrifugal cicatricial alopecia
- Stem cells and aberrant signaling of molecular systems in skin aging
- The Transmembrane Serine Protease HAT-like 4 Is Important for Epidermal Barrier Function to Prevent Body Fluid Loss
- Noncontraceptive benefits of the estradiol valerate/dienogest combined oral contraceptive: a review of the literature
- AROMA THERAPY: AN ART OF HEALING
- Current aspects of polycystic ovary syndrome: A literature review
- Indian Gooseberry (<i>Emblica officinalis</i>Gaertn.)
- Hormones
- Estrogen modulates mesenchymal-epidermal interactions in the adult nipple
- Glycolytic Dysfunction in Granulosa Cells and Its Contribution to Metabolic Dysfunction in Polycystic Ovary Syndrome