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The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

The technology can help diagnose and subtype autoimmune diseases by identifying specific autoantibodies.

Human hair proteins, especially keratins, can protect cells from oxidative stress in lab settings.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Wnt7a protein is crucial for development and tissue maintenance and plays varying roles in diseases and potential treatments.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

Blocking YAP/TAZ could be a new way to treat skin cancer.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Blood tests for tumor cells could improve prostate cancer diagnosis and treatment; hair loss severity linked to a gene affecting prostate conditions.

Vitamin D and its receptor may help prevent skin cancer.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Inactive hair follicle stem cells help prevent skin cancer.

The transgene likely activated an oncogene or interrupted a tumor suppressor gene, causing melanoma in mice.

NDRG1 protein helps infantile hemangioma, a common infant tumor, to grow, and its mismanagement by FOXO1 protein plays a big role in causing the tumor.

Researchers discovered potential origins and new treatments for skin cancer, including biomarkers for melanoma and therapies that reduce tumor growth.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

Using protein degradation to fight cancer drug resistance shows promise but needs more precise targeting and fewer side effects.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.

KLF4 is important for maintaining stem cells and has potential in cancer treatment and wound healing.

The article concludes that while we understand a lot about how melanocytes age and how this can prevent cancer, there are still unanswered questions about certain pathways and genes involved.

The document concludes that the immune-inhibitory environment of the hair follicle may prevent melanoma development.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

New markers and pathways have been found in skin tumors, helping better understand and diagnose them.

VEGF helps squamous cell carcinoma grow in ways beyond just blood vessel formation.

High levels of ST14 and TMEFF1 proteins in ovarian cancer are linked to worse patient outcomes and may be a new treatment target.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.