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A new keratin, hK6irs1, is found in all layers of the hair follicle's inner root sheath.

Hair growth can be stimulated by combining certain skin cells, which can rejuvenate old cells and cause them to specialize in hair follicle creation.

The project created a standardized system for classifying skin lesions in lab rats and mice.

TGM3 is important for skin and hair structure and may help diagnose cancer.

MMP-9 is essential for proper hair canal formation.

A specific genetic change in the KRT71 gene causes a hair loss condition in Hereford cattle.

Some leukemia treatments can cause skin reactions similar to keratosis pilaris.

Hair matrix cysts are rare skin nodules with unique features, often needing surgical removal.

Tigers had a skin condition causing hair loss and inflammation, but the cause is unknown and treatment didn't work.

The C-terminal tail of AHF/trichohyalin is essential for organizing keratin filaments in keratinocytes.

The skin is a large organ that plays a role in the immune system.

Trichohyalin is also found in the outer layers of normal human skin.

Tongue cancer cells show more plectin-1 than non-cancer cells, but both have similar levels of trichohyalin.

Cathepsin L is essential for normal hair growth and development.

Wool follicles are complex, involving interactions between different cell types and structures.

Enzymes called PADIs play a key role in hair growth and loss.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

New protein changes may be involved in the immune attack on hair follicles in alopecia areata.

PAD3 plays a key role in hair and skin protein structure and may be linked to skin diseases.

Mouse profilaggrin helps in skin cell differentiation and may be involved in calcium signaling.

A gene mutation causes early keratinocyte maturation leading to hair loss in Olmsted syndrome.

The document concludes that alopecia has significant social and psychological effects, leading to a market for hair loss treatments.

The article explains the genetic causes and symptoms of various hair disorders and highlights the need for more research to find treatments.

Different populations have distinct hair structures related to their ancestry.

Some mutant mice have hair with abnormal cross-linking, mainly in the cuticle, not affecting other hair parts.

People with alopecia areata have higher levels of RBP4 protein and antibodies against it.

Understanding hair growth involves complex interactions between molecules and could help treat hair disorders.

The document concludes that accurate diagnosis of ichthyosis is crucial for treatment and genetic advice, and ongoing research is needed for better therapies.

The article concludes that the wool follicle is a valuable model for studying tissue interactions and has potential for genetic improvements in wool production.

The document concludes that hair is complex, with a detailed growth cycle, structure, and clinical importance, affecting various scientific and medical fields.