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Research 31–60 of 1000+
- Targeted expression of SV40 T antigen in the hair follicle of transgenic mice produces an aberrant hair phenotype
- Growth factors and cytokines in hair follicle development and cycling: recent insights from animal models and the potentials for clinical therapy
- Ethanol extract of Sinsun-yukza-hwan, a Korean medicinal prescription, promotes hair growth in C57BL/6 mice, an alopecia animal model
- The role of Wnt and Shh signaling systems in noggin-induced tumorigenesis
- Morpho-Regulation of Ectodermal Organs
- 2063-LB: Transgenic Erythropoietin Overexpression Inhibits Dermal Fat Formation and HIF1a in the Skin and Causes Telogen Effluvium in Mouse Pups during the First Hair Cycle
- Overexpression of spermidine/spermine N1-acetyltransferase under the control of mouse metallothionein I promoter in transgenic mice: evidence for a striking post-transcriptional regulation of transgene expression by a polyamine analogue
- Overexpression of insulin-like growth factor-1 induces hyperplasia, dermal abnormalities, and spontaneous tumor formation in transgenic mice
- Development of Heritable Melanoma in Transgenic Mice
- A 5'-upstream region of a bovine keratin 6 gene confers tissue-specific expression and hyperproliferation-related induction in transgenic mice.
- The Human Papillomavirus Type 11 Upstream Regulatory Region Triggers Hair-Follicle-Specific Gene Expression in Transgenic Mice
- Skin-specific transgenic overexpression of ovine β-catenin in mice
- Mutant laboratory mice with abnormalities in hair follicle morphogenesis, cycling, and/or structure: An update
- High‐Sulfur Protein Gene Expression in a Transgenic Mouse
- Relation of Skin Polyamines to the Hairless Phenotype in Transgenic Mice Overexpressing Spermidine/Spermine N1-Acetyltransferase
- Smad4-dependent desmoglein-4 expression contributes to hair follicle integrity
- Transgenic Kallikrein 14 Mice Display Major Hair Shaft Defects Associated with Desmoglein 3 and 4 Degradation, Abnormal Epidermal Differentiation, and IL-36 Signature
- A Mouse Model of Androgenetic Alopecia
- Hairless triggers reactivation of hair growth by promoting Wnt signaling
- Impaired wound healing in transgenic mice overexpressing the activin antagonist follistatin in the epidermis
- Activation of Polyamine Catabolism Profoundly Alters Tissue Polyamine Pools and Affects Hair Growth and Female Fertility in Transgenic Mice Overexpressing Spermidine/SpermineN 1-Acetyltransferase
- Directed Expression of Keratin 16 to the Progenitor Basal Cells of Transgenic Mouse Skin Delays Skin Maturation
- Epigen Transgenic Mice Develop Enlarged Sebaceous Glands
- Hair-specific expression of chloramphenicol acetyltransferase in transgenic mice under the control of an ultra-high-sulfur keratin promoter.
- Premature aging and cancer development in transgenic mice lacking functional CYLD
- Restoration of Spermatogenesis and Male Fertility Using an Androgen Receptor Transgene
- Krt6a-Cre Transgenic Mice Direct LoxP-Mediated Recombination to the Companion Cell Layer of the Hair Follicle and Following Induction by Retinoic Acid to the Interfollicular Epidermis
- One Transgene: Two Outcomes
- One Transgene: Two Outcomes
- Reversing wrinkled skin and hair loss in mice by restoring mitochondrial function