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The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

Seasonal changes affect gene activity linked to hair growth in Angora goats.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

FGF5 spliceosomes inhibit rabbit hair growth by affecting gene expression.

PRP is a promising treatment for hair loss.

Type 2 diabetic stem cells can still help heal wounds effectively.

Ginsenoside Rb1 may help remodel hypertrophic scars effectively at a dose of 0.56 mg.

TGF-alpha is present in sheep and ferret skin and may affect hair growth without directly stimulating cell proliferation.

The research identified proteins that change as goat hair follicles begin to form, helping to understand how cashmere grows.

Certain genes and pathways are linked to the production of finer and denser wool in Hetian sheep.

The 2004 hair research meeting presented new findings on hair cell differentiation, genetic factors in hair loss, hair pigmentation, and potential targeted therapies.

Scientists found key proteins and genes that affect skin and hair health, and identified potential new treatments for hair loss, skin disorders, and wound healing.

Fibromodulin might help reduce scarring if increased in adult wounds like in fetal skin that heals without scars.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

The lentiviral array can monitor and predict gene activity during stem cell differentiation.

Blocking EGFR in skin cells doesn't majorly increase inflammation markers.

A new wound dressing with p-Coumaric acid helps heal diabetic wounds faster by reducing inflammation and promoting skin repair.

Hyaluronic acid and polycaprolactone improve skin regeneration, with polycaprolactone having a stronger effect on healing and tissue repair.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

People with severe Alopecia Areata have higher levels of TGF-β1, which may play a role in the condition.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

The document concludes that recent research has improved understanding of skin diseases and the balance between cell growth and differentiation in the epidermis.

TNF family proteins are crucial for the development of skin features like hair, teeth, and mammary glands.

Implanting hair-follicle stem cells in mice brains helped repair brain bleeding and reduced brain inflammation.

A specific genetic marker is linked to male pattern baldness in Han Chinese men.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

TGF-β is crucial for controlling stem cell behavior and changes in its signaling can lead to diseases like cancer.

Current and future treatments for alopecia areata focus on immunosuppression, immunomodulation, and protecting hair follicles.