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Research 481–510 of 638
- p63 in skin homeostasis and disease: molecular mechanisms and therapeutic potentials
- Design, synthesis and anti-tumor evaluation of novel thiohydantoin congeners as androgen receptor antagonists with in vivo study.
- Molecular mechanisms of Y chromosome loss and <i>UTY</i> gene activity
- AP-2α/AP-2β transcription factors are key regulators of epidermal homeostasis
- Calcineurin/Nuclear Factor of Activated T-Cell Pathway in Cutaneous Squamous Cell Carcinoma
- Role of polymorphism of the androgen receptor gene and non-random X chromosome inactivation in the genesis of androgenic alopecia in women of childbearing potential
- Analyse de la régulation du gène Hairless et identification des voies de signalisation affectées chez les mutants «bald Mill Hill».
- Off-label uses of retinoids in dermatology
- p63 expression in normal skin and usual cutaneous carcinomas
- Conversion of C57Bl/6 mice from a tumor promotion-resistant to a -sensitive phenotype by enhanced ornithine decarboxylase expression
- Potential of Macroalgae for Anti Alopecia: A Systematic Review
- Conversion of C57Bl/6 mice from a tumor promotion–resistant to a –sensitive phenotype by enhanced ornithine decarboxylase expression
- Epidermal homeostasis: a balancing act of stem cells in the skin
- Epithelial Stem Cells: Turning over New Leaves
- Fibroblasts: Origins, definitions, and functions in health and disease
- Transgenic mice provide new insights into the role of TGF-alpha during epidermal development and differentiation.
- New aspects of vitamin D metabolism and action — addressing the skin as source and target
- c-Myc activation in transgenic mouse epidermis results in mobilization of stem cells and differentiation of their progeny
- Polymorphism of the Androgen Receptor Gene is Associated with Male Pattern Baldness
- Understanding, recognizing, and managing toxicities of targeted anticancer therapies
- Matriptase: Potent Proteolysis on the Cell Surface
- Genetic Disorders and Defects in Vitamin D Action
- Atrichia Caused by Mutations in the Vitamin D Receptor Gene is a Phenocopy of Generalized Atrichia Caused by Mutations in the Hairless Gene
- Regenerating the skin: a task for the heterogeneous stem cell pool and surrounding niche
- Resting no more: re‐defining telogen, the maintenance stage of the hair growth cycle
- Local circadian clock gates cell cycle progression of transient amplifying cells during regenerative hair cycling
- Dermal fibroblast in cutaneous development and healing
- Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets
- Concise Review: Wnt Signaling Pathways in Skin Development and Epidermal Stem Cells
- Epidermal Stem Cells Are Defined by Global Histone Modifications that Are Altered by Myc-Induced Differentiation