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Acne is linked to inflammation and insulin resistance, and is associated with various syndromes that require different treatments.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

UK medical students lack dermatology education, liver biopsy patients with risk factors show more fibrosis, and certain fungi resist drugs due to melanin; genetics may influence female hair loss.

The workshop highlighted the genetic links and psychological impacts of hair loss and skin disorders.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Circadian rhythms are crucial for stem cell function and tissue repair, and understanding them may improve aging and regeneration treatments.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Cicatricial alopecia, a permanent hair loss condition, is mainly caused by damage to specific hair follicle stem cells and abnormal immune responses, with gene regulator PPAR-y and lipid metabolism disorders playing significant roles.

TLR3 signaling enhances the immunosuppressive properties of human periodontal ligament stem cells.

FOL-026 peptide can help repair blood vessels and promote growth, offering potential treatment for vascular diseases.

Natural compounds, especially Withaferin-A, may help treat post-COVID-19 complications, but some may have side effects.

2011 dermatological research found new skin aging markers, hair loss causes, skin defense mechanisms, and potential for new treatments.

Short-chain fatty acids from *Cutibacterium acnes* cause skin inflammation, contributing to acne.

The article suggests that targeting specific immune pathways could help control and treat the skin disease hidradenitis suppurativa.

Acne is caused by multiple factors including oil production, bacteria, inflammation, and possibly diet and environment.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Mutations in the glucocorticoid receptor gene cause reduced sensitivity to glucocorticoids and may lead to poor response to treatment.

Targeted cancer therapies often cause skin problems that need careful management to improve patient quality of life and treatment success.

Hair can regrow in large wounds through a process similar to how hair forms in embryos, and understanding this could lead to new treatments for hair loss or scarring.

Erlotinib causes skin inflammation through IL-1, which can be reduced by anakinra.

Botryococcus terribilis Ethanol Extract may reduce inflammation by changing gene expression in cells.

Alopecia areata severity and treatment response are linked to specific cytokine levels.

Eating a high-fat fish oil diet caused mice to lose hair due to a specific immune cell activity in the skin linked to a protein called E-FABP.

Personalized medicine in dermatology uses molecular biomarkers to improve diagnosis and treatment but needs further advancements for practical use.

We now understand more about what causes acne and this could lead to better, more personalized treatments.

Melasma's causes include genetics, sun exposure, hormones, and oxidative stress, and understanding these can help create better treatments.

Hair loss in men may be caused by inflammation triggered by hormones, and treatments reducing inflammation could potentially promote hair growth.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Notch signalling helps skin cells differentiate and prevents tumors.