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Research 721–750 of 1000+
- Hair aging and anti-aging
- Benefit-Risk Assessment of GLP-1 Receptor Agonists: Implications for Dermatologists and Plastic Surgeons
- Factors Associated with Severity of Alopecia Areata
- The Biology of Hair Follicles
- Anatomy and Physiology of the Skin
- Deletion of Deoxyribonucleic Acid Binding Domain of the Vitamin D Receptor Abrogates Genomic and Nongenomic Functions of Vitamin D
- Androgen Receptor Antagonists (Antiandrogens) Structure-Activity Relationships
- Evaluation and treatment of male and female pattern hair loss
- Frontal fibrosing alopecia in postmenopausal women
- Decreased Serum Ferritin is Associated With Alopecia in Women
- Obesity and the skin
- Drug-Induced Skin, Nail and Hair Disorders
- Hair Follicle Immune Privilege Revisited: The Key to Alopecia Areata Management
- Selenoproteins Are Essential for Proper Keratinocyte Function and Skin Development
- Current evaluation of amenorrhea
- Updated Treatment for Acne: Targeted Therapy Based on Pathogenesis
- Tumour regression predicts higher risk of sentinel node involvement in thin cutaneous melanomas
- The control of hair growth: An overview
- Selenium or No Selenium- That Is the Question in Tumor Patients: A New Controversy
- Cutaneous and Mucosal Manifestations Associated with Celiac Disease
- Premature graying of hair
- Hair: What is New in Diagnosis and Management?
- Nanotechnology-Based Cosmetics for Hair Care
- Polycystic Ovary Syndrome and Its Differential Diagnosis
- Analysis of 1,25-Dihydroxyvitamin D3 Receptors (VDR. in Basal Cell Carcinomas
- Comprehensive analysis of circRNAs from cashmere goat skin by next generation RNA sequencing (RNA-seq)
- Lymphoid Enhancer-binding Factor-1 (LEF1) Interacts with the DNA-binding Domain of the Vitamin D Receptor
- Assessment of the usefulness of dihydrotestosterone in the diagnostics of patients with androgenetic alopecia
- Pathogenesis and treatment options for chemotherapy‐induced alopecia: a systematic review
- Human hair follicles operate an internal Cori cycle and modulate their growth via glycogen phosphorylase