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Research 22
- Tgm2/Gh, Gbx1 and TGF-beta are involved in retinoic acid-induced transdifferentiation from epidermis to mucosal epithelium
- Retinoic Acid-Induced Epidermal Transdifferentiation in Skin
- 110 Transglutaminase 2 is a potential regulator of sebocyte maturation
- Helminth protein enhances wound healing by inhibiting fibrosis and promoting tissue regeneration
- Targeted Inactivation of Gh/Tissue Transglutaminase II
- Self-organization process in newborn skin organoid formation inspires strategy to restore hair regeneration of adult cells
- CD201+ fascia progenitors choreograph injury repair
- γδ17 T cell-stromal networks modulate matrix composition and vascularity in foreign body response
- IL-17a induces age-related olfactory dysfunction by impairing regeneration and promoting respiratory metaplasia in mice
- <i>Malassezia</i> and the Asian menopausal skin
- Wasabi leaf extract changes the expression levels of cytokine related genes in dermal papilla cells, which identified by the whole transcriptome
- AP-2α/AP-2β transcription factors are key regulators of epidermal homeostasis
- Mechanical Stretching Can Modify the Papillary Dermis Pattern and Papillary Fibroblast Characteristics during Skin Regeneration
- Hair Follicular Expression and Function of Group X Secreted Phospholipase A2 in Mouse Skin
- The Aldo-Keto Reductase AKR1B10 Is Up-Regulated in Keloid Epidermis, Implicating Retinoic Acid Pathway Dysregulation in the Pathogenesis of Keloid Disease
- Transglutaminase 3: The Involvement in Epithelial Differentiation and Cancer
- Cyanidin 3-O-arabinoside suppresses DHT-induced dermal papilla cell senescence by modulating p38-dependent ER-mitochondria contacts
- Pathological Mechanisms Involved in Epidermolysis Bullosa Simplex: Current Knowledge and Therapeutic Perspectives
- Exploring the Complexities of TGF-beta Signaling in Keloids: Beyond the Classical Smad Pathway
- Fibroblast Lineage Switching as the Developmental Origin of Scarring and Target for Regenerative Healing
- Fibroblast Lineage Switching as the Developmental Origin of Scarring and Target for Regenerating Healing
- Human Fibroblast‐Derived Matrix Hydrogel Accelerates Regenerative Wound Remodeling Through the Interactions with Macrophages