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All-trans-retinoic acid stops mink hair growth by affecting cell growth and causing cell death.

A peptide called DPS-1 helps human scalp cells grow and stimulates hair growth in mice.

Certain growth factors significantly affect hair loss in women with telogen effluvium.

Hair follicle stem cell transplants can reverse liver cirrhosis by blocking harmful cell activation.

Winter provides the best fur quality for Rex rabbits due to seasonal changes in specific signaling pathways.

TGF-β signaling is essential for new hair growth after a wound.

YH0618 helps reduce chemotherapy-induced hair loss by targeting specific proteins and pathways.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

Muscle around hair follicles controls hair loss by releasing a signal that causes cell death.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

A parasite molecule can speed up skin healing and reduce scarring.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Endoglin is important for proper hair growth cycles and stem cell activation in hair follicles.

TGF-β is found in hair follicles and may cause hair loss in alopecia areata.

ROS may affect hair loss related to hormones.

Activins and follistatins, part of the TGFβ family, are crucial for hair follicle development and skin health, affecting growth, repair, and the hair cycle.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

Smad2/3-dependent TGF-β signaling increases during wound healing.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Prolactin and TGF-β receptor blockers might help treat hair loss.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Artificial skin development has challenges, but new materials and understanding cell behavior could improve tissue repair. Also, certain growth factors and hydrogel technology show promise for advanced skin replacement therapies.

New effective scar treatments are urgently needed due to the current options' limited success.

Different fibroblasts play key roles in skin healing and scarring.

Capsaicin, found in chili peppers, can slow down hair growth by affecting skin cells and hair follicles.

Fibromodulin might help reduce scarring if increased in adult wounds like in fetal skin that heals without scars.

Stem cell niches support, regulate, and coordinate stem cell functions.

Understanding hair growth involves complex interactions between molecules and could help treat hair disorders.