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A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

A parasite molecule can speed up skin healing and reduce scarring.

People with severe Alopecia Areata have higher levels of TGF-β1, which may play a role in the condition.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Endoglin is important for proper hair growth cycles and stem cell activation in hair follicles.

HPH-15, a new compound, effectively reduces skin fibrosis in experiments without causing harm.

4-O-methylhonokiol from Magnolia Officinalis may promote hair growth by interfering with TGF-β1 effects in skin cells.

TGF-β is found in hair follicles and may cause hair loss in alopecia areata.

TGFβ-2 may cause hair loss in androgenetic alopecia.

ROS may affect hair loss related to hormones.

Low oxygen levels affect the behavior of certain proteins in human skin cells.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Targeting TGFβ can improve skin immunity in older people.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Activins and follistatins, part of the TGFβ family, are crucial for hair follicle development and skin health, affecting growth, repair, and the hair cycle.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Smad2/3-dependent TGF-β signaling increases during wound healing.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

TGF-alpha is present in sheep and ferret skin and may affect hair growth without directly stimulating cell proliferation.

Male hormones can cause hair loss, but treatments like Minoxidil and Finasteride can help, and targeting TGF-B1 could be a future solution.

Prolactin and TGF-β receptor blockers might help treat hair loss.

Male pattern baldness is caused by certain cells in hair follicles and could potentially be treated by targeting a specific growth factor, TGF-β1.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.