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ADSC-Exos with miR-122-5p can help treat hair loss by promoting hair growth.

ROS may affect hair loss related to hormones.

Low oxygen levels affect the behavior of certain proteins in human skin cells.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

ECM1-modified stem cells can effectively treat liver cirrhosis.

TGF-β is crucial for tissue repair and can cause diseases if not properly regulated.

Targeting TGFβ can improve skin immunity in older people.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Activins and follistatins, part of the TGFβ family, are crucial for hair follicle development and skin health, affecting growth, repair, and the hair cycle.

LHX2 is essential for hair follicle development, controlled by NF-κB and TGFβ2 signaling.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Smad2/3-dependent TGF-β signaling increases during wound healing.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

The research suggests that p63 and TGF-β1 may help determine tumor type and malignancy in hair follicle and sebaceous tumors.

TGF-alpha is present in sheep and ferret skin and may affect hair growth without directly stimulating cell proliferation.

Male hormones can cause hair loss, but treatments like Minoxidil and Finasteride can help, and targeting TGF-B1 could be a future solution.

Prolactin and TGF-β receptor blockers might help treat hair loss.

Male pattern baldness is caused by certain cells in hair follicles and could potentially be treated by targeting a specific growth factor, TGF-β1.

TGF-β signaling is essential for new hair growth after wounds.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Botulinum toxin affects hair follicles, inhibiting TGF-B1 secretion; more research ongoing.

Fibroblasts are crucial for tissue repair and inflammation, and understanding them can help treat fibrotic diseases.

The conclusion is that certain cell interactions and signals are crucial for hair growth and regeneration.

Melanocyte stem cells in hair follicles are key for hair color and could help treat greying and pigment disorders.

The document concludes that recent research has improved understanding of skin diseases and the balance between cell growth and differentiation in the epidermis.