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A new wound dressing with p-Coumaric acid helps heal diabetic wounds faster by reducing inflammation and promoting skin repair.

People with severe Alopecia Areata have higher levels of TGF-β1, which may play a role in the condition.

SH-MSCs gel reduced IL-6 and increased TGF-β, suggesting it could treat alopecia.

Muscle around hair follicles controls hair loss by releasing a signal that causes cell death.

TGF-β is found in hair follicles and may cause hair loss in alopecia areata.

ADSC-Exos with miR-122-5p can help treat hair loss by promoting hair growth.

TGF-β2 helps activate hair follicle stem cells by counteracting BMP signals.

P144® improves hypertrophic scars by reducing size and thickness and increasing elasticity.

A peptide called DPS-1 helps human scalp cells grow and stimulates hair growth in mice.

5-Bromo-3,4-dihydroxybenzaldehyde could potentially help hair growth by activating certain cell pathways and inhibiting others.

4-O-Methylhonokiol helps protect skin cells from growth-stopping effects of a protein by regulating growth-related pathways.

Two microRNAs affect hair follicle development in sheep by targeting specific genes.

A new peptide from ginseng berries may help prevent hair loss by promoting cell growth and reducing stress damage.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.

A parasite molecule can speed up skin healing and reduce scarring.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

Hair follicle stem cell transplants can reverse liver cirrhosis by blocking harmful cell activation.

Low oxygen levels affect the behavior of certain proteins in human skin cells.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

TGF-β signaling is crucial for stem cell maintenance, differentiation, and has implications for cancer treatment.

Targeting TGFβ can improve skin immunity in older people.

All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

The conclusion is that Fgf18 and Tgf-ß signaling could be targeted for hair loss treatments.

Wnt/β-catenin and TGF-β pathways affect hair loss, and activating Wnt/β-catenin could be a potential treatment.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Prolactin and TGF-β receptor blockers might help treat hair loss.

Higher TGF-β signaling may increase skin cancer risk in organ transplant recipients.

The mouse model shows potential for understanding and improving scarless wound healing, and Wnt-4 and TGF-β1 play a role in wound healing and scar formation.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.