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The document concludes that pregnancy and cancer share immune evasion tactics, but more research is needed before using checkpoint blockade immunotherapy in pregnant cancer patients to avoid harm to the placenta.

Human prenatal skin develops an immune network early on that helps with skin formation and healing without scarring.

Understanding the immune-related causes of Alopecia Areata has led to potential treatments like JAK inhibitors.

Flutamide caused damage to male rat reproductive organs and may affect fertility.

Interferons are effective for some skin conditions and cancers, but can have side effects and need more research for optimal use.

Alopecia areata is an autoimmune condition causing hair loss, influenced by genetics, stress, and diet, and may be prevented by a high soy oil diet.

Methotrexate is a key, cost-effective drug for skin conditions, but requires careful monitoring for side effects.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Visible light can damage skin and most sunscreens don't block it well; more research is needed on its effects and protection methods.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Pure carbon nanotubes are safe for mice, but impure ones cause immune issues and hair loss.

Keratinocytes help heal skin wounds by interacting with immune cells and producing substances that kill pathogens.

Scientists found cells in hair that are key for growth and color.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

The skin has a complex immune system that is essential for protection and healing, requiring more research for better wound treatment.

Fibroblast Growth Factors (FGFs) are important for tissue repair and regeneration, influencing cell behavior and other factors involved in healing, and are crucial in processes like wound healing, bone repair, and hair growth.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

EdnrB signaling helps melanocyte stem cells regenerate and could be targeted to treat pigmentation issues.

Imiquimod cream activates hair follicle stem cells and causes early hair growth by changing immune cells and certain protein expressions.

Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Apple extract called procyanidin B-2 was found to greatly increase hair growth.

Old treatments for other skin conditions showed promise for hair regrowth in mice with a hair loss condition.

Stress turns hair white by depleting color-giving cells in hair follicles through a specific neurotransmitter related to the body's stress response.

Stem cells are crucial for skin repair and new treatments for chronic wounds.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

Loss of a specific protein in skin cells causes symptoms similar to psoriasis.