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All-trans retinoic acid causes hair loss by increasing TGF-β2 in hair follicle cells.

The document concludes that understanding the genes and pathways involved in hair growth is crucial for developing treatments for hair diseases.

Cholesterol sulfate buildup due to a genetic mutation disrupts the skin barrier, leading to the scaling skin seen in X-linked ichthyosis.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

The Foxn1 gene mutation causes hairlessness and immune system issues, and understanding it could lead to hair growth disorder treatments.

Mutant mice help researchers understand hair growth and related genetic factors.

New methods for skin and nerve regeneration can improve healing and feeling after burns.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Activin helps skin growth and healing mainly through stromal cells and affects keratinocytes based on its amount.

COVID-19 can cause various skin lesions, which may result from the virus and immune response, and are not directly linked to illness severity.

TPA is about 50 times more effective at promoting tumors than MZ.

YAP and TAZ proteins are necessary for the development of two types of skin cancer.

Targeting the skin's endocannabinoid system could help treat skin disorders.

Aging mice have slower hair regeneration due to changes in signal balance, but the environment, not stem cell loss, controls this, suggesting treatments could focus on environmental factors.

Mice lacking a key DNA methylation enzyme in skin cells have a lower chance of activating stem cells necessary for hair growth, leading to progressive hair loss.

Wnt signaling is crucial for skin and hair development and its disruption can cause skin tumors.

Keratin proteins are crucial for healthy skin, but mutations can cause skin disorders with no effective treatments yet.

The study identified and characterized new keratin genes linked to hair follicles and epithelial tissues.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

Acne is caused by genetics, diet, hormones, and bacteria, with treatments not yet curative.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Anti-acne medications may work by reducing the activity of a protein involved in acne development.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Prolactin affects the production of different keratins in human hair, which could lead to new treatments for skin and hair disorders.

Vitiligo causes white skin patches and is linked to autoimmune issues.

Epidermal stem cells could lead to new treatments for skin and hair disorders.

Notch-related genes play a key role in the development and cycling of hair follicles.

RANK-RANKL signaling is essential for hair growth and skin health.

Applying a specific inhibitor lightens skin and hair color.

Two-photon microscopy helps observe hair follicle stem cell behaviors in mice.