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The gel with icariin speeds up wound healing, reduces scarring, and helps hair growth by controlling BMP4 signaling. It also reduces inflammation and improves wound quality in mice, adapts to different wound shapes, and gradually releases icariin to aid healing. It also prevents too much collagen and myofibroblast formation during skin healing.

Hair and skin healing involve complex cell interactions controlled by specific molecules and pathways, and hair follicle cells can help repair skin wounds.

Eyelid cells share signaling components but differ in pathway activity.

BMPR1a-ECD reduces wrinkles much more effectively than retinoic acid.

Researchers created a mouse cell line to study hair growth and test hair growth drugs.

Blocking miR-27a increases sheep hair follicle stem cell growth and decreases cell death, which could help improve wool quality and treat hair loss.

Communication between blood vessel and hair follicle cells decreases with age, affecting hair growth and blood vessel formation.

TLR2 helps control hair growth and regeneration, and its reduction with age or obesity can impair hair growth.

miR-29a-5p prevents the formation of early hair structures by targeting a gene important for hair growth and is regulated by a complex network involving lncRNA627.1.

Activin A and follistatin control when ear hair cells form in mice.

Activin A promotes ear hair cell development, while follistatin delays it.

Low-Level Laser Therapy might be an effective alternative hair loss treatment for some people.

Low-Level Laser Therapy might be a good alternative for hair loss when other treatments fail, but its effectiveness varies and more research is needed.

Targeting CXXC5 and GSK-3β may help treat male pattern baldness.

TGF-β1 increases androgen receptor activity in hair loss, but Hic-5/ARA55 can counter this effect.

Keratinocytes show more TGF-β system activity and collagen production as they age, which might affect wound scarring.

Smad2/3-dependent TGF-β signaling increases during wound healing.

Lower SMAD2/3 activation predicts more severe skin cancer.

All-trans-retinoic acid stops mink hair growth by affecting cell growth and causing cell death.

Hair follicle stem cell transplants can reverse liver cirrhosis by blocking harmful cell activation.

YH0618 helps reduce chemotherapy-induced hair loss by targeting specific proteins and pathways.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

HPH-15, a new compound, effectively reduces skin fibrosis in experiments without causing harm.

The BMP/Smads pathway and Id2 gene control hair follicle stem cells, affecting their rest and growth phases.

ADSC-Exos with miR-122-5p can help treat hair loss by promoting hair growth.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

Keloid scars may form due to changes in skin cell characteristics and specific protein signaling.

MicroRNA-21 could help diagnose and treat skin fibrosis.

Endoglin is crucial for proper hair growth cycles and stem cell activation in mice.