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ADSC-derived extracellular vesicles show promise for skin and hair regeneration and wound healing.

Sox2-positive dermal papilla cells have unique characteristics and contribute more to skin and hair follicle formation than Sox2-negative cells.

Fat cells help recruit healing cells and build skin structure during wound healing.

Human skin cells can create new hair follicles when transplanted into mice.

GDNF helps grow hair and heal skin wounds by acting on hair stem cells.

Skin cells show flexibility in healing wounds and forming tumors, with potential for treating hair disorders and chronic ulcers.

Skin patterns form through molecular signals and genetic factors, affecting healing and dermatology.

The experiment showed that human skin grown in the lab started to form early hair structures when special cell clusters were added.

Skin patterns are formed by simple reaction-diffusion mechanisms.

Increased DHT formation in skin can cause hair loss, acne, and excessive hair growth, and antiandrogens might treat these conditions.

Differentiated fibroblasts regenerate hair follicles better than undifferentiated ones.

Deleting the CRIF1 gene in mice disrupts skin and hair formation, certain proteins affect hair growth, a new compound may improve skin and hair health, blood cell-derived stem cells can create skin-like structures, and hair follicle stem cells come from embryonic cells needing specific signals for development.

Different scaffold patterns improve wound healing and immune response in mouse skin, with aligned patterns being particularly effective.

Porcine acellular dermal matrix treatment helps wounds heal faster and reduces scarring by affecting Jag1 in skin stem cells.

Dermal papilla cells help wounds heal better and can potentially grow new hair.

TCF/Lef1 is essential for skin barrier function by regulating lipid metabolism.

New molecules involved in skin and hair growth were identified, improving understanding and future treatments.

PEVs effectively deliver minoxidil through skin.

The hydrogel helps reduce scarring and improve wound healing by releasing salvianolic acid B in acidic conditions.

Skin lesions in Carney Complex are caused by a gene change in some skin cells that leads to increased pigmentation and may lead to tumors.

Many popular skincare products claim to prevent aging but lack strong evidence to prove their effectiveness and safety.

The gel with icariin speeds up wound healing, reduces scarring, and helps hair growth by controlling BMP4 signaling. It also reduces inflammation and improves wound quality in mice, adapts to different wound shapes, and gradually releases icariin to aid healing. It also prevents too much collagen and myofibroblast formation during skin healing.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.

Hormones and stretching both needed for nipple area skin growth in mice.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

iPSC-derived stem cells on a special membrane can help repair full-thickness skin defects.

Cornification is how skin cells die to form the protective outer layer of skin, hair, and nails.

Fat cells are important for healthy skin, hair growth, and healing, and changes in these cells can affect skin conditions and aging.

Dermal papillae cells, important for hair growth, come from multiple cell lines and can be formed by skin cells, regardless of their origin or hair cycle phase. These cells rarely divide, but their ability to shape tissue may contribute to their efficiency in inducing hair growth.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.