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Rat whisker cells can help turn other cells into nerve cells and might be used to treat brain injuries or diseases.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Type I interferons play a key role in the development of various skin diseases.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

Natural ingredients in cosmeceuticals are beneficial for skin and hair health with few side effects.

Mutant mice help researchers understand hair growth and related genetic factors.

Human hair follicles produce and respond to erythropoietin, helping protect against stress.

Apoptosis helps shape hair growth and prepares the skin for hair to emerge.

Phosphatidic acid may help hair grow by affecting cell growth pathways.

Cold atmospheric plasma may speed up wound healing and control infections.

Cathepsin L is essential for normal hair growth and development.

Scientists have found a way to create hair follicles from skin cells of newborn mice, which can grow and cycle naturally when injected into adult mouse skin.

Epithelial-mesenchymal interactions control hair growth cycles through specific molecular signals.

Hair loss in androgenic alopecia patients is linked to changes in certain genes that control cell growth and death.

Electrical epilation damages hair follicles and surrounding skin, likely preventing hair regrowth.

Restoring mitochondrial function in mice reversed their skin wrinkling and hair loss.

Activin A helps heal skin wounds and protects the brain after injury.

Caspase-7 has functions in skin and hair that are not related to cell death.

Blocking mTORC1 activity with rapamycin could help increase hair pigmentation and growth, potentially reversing gray hair.

Proper cell death regulation is crucial for normal hair follicle regeneration and skin remodeling.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

EGFR is essential for normal hair development and follicle differentiation.

Inflammation in psoriasis may trigger antimicrobial peptides and cell death.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Immune checkpoint inhibitors can cause skin issues but are linked to better cancer outcomes.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

TAF4 is important for skin cell growth and helps prevent skin cancer in mice.