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Topical ceramide treatment partially improves the skin condition in Jack Russell Terriers with a genetic skin disorder.

The skin is a complex barrier for drug penetration, but understanding its structure and interactions can improve drug delivery methods.

ACBP is essential for healthy skin and fur by maintaining the skin's barrier function.

PPARγ is essential for maintaining healthy skin, controlling inflammation, and ensuring proper skin barrier function.

TCDD speeds up skin barrier formation by increasing certain gene expressions.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.

Fatty acid transport protein 4 is essential for skin and hair development.

Certain gene mutations can weaken the skin barrier and, when combined with environmental factors, lead to eczema and severe itching.

Skin aging is caused by stem cell damage and can potentially be delayed with treatments like antioxidants and stem cell therapy.

IL-1 family cytokines are crucial for skin defense and healing, but their imbalance can cause skin diseases.

Inhibiting AP1 in mice skin causes structural changes and weakens the skin barrier.

AcSDKP may help prevent skin and hair aging and promote their growth.

A new non-invasive method can analyze skin mRNA to understand skin diseases better.

Skin aging reflects overall body aging and can indicate internal health conditions.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

ATP-dependent chromatin remodeling is crucial for skin development and stem cell function.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

The nucleus is key in controlling skin growth and repair by coordinating signals, gene regulators, and epigenetic changes.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

Keratosis pilaris is often linked to genetic mutations and causes skin and hair abnormalities, regardless of those mutations.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Removing the Crif1 gene in mouse skin disrupts skin balance and hair growth.

The document concludes that adult mammalian skin contains multiple stem cell populations with specific markers, important for understanding skin regeneration and related conditions.

A gene called Gk5 controls lipid production in the skin and affects hair growth.

Hair follicles are essential for skin health, aiding in hair growth, wound healing, and immune function.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Topical treatments can deliver active molecules to skin stem cells, potentially helping treat skin and hair disorders, including skin cancers and hair loss.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.