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Different small areas within hair follicles send specific signals that control what type of cells stem cells become.

Patients with systemic lupus erythematosus and hair loss had non-scarring alopecia with fewer hair follicles, and direct immunofluorescence did not help identify lupus.

The research created a detailed map of skin cells, showing that certain cells in basal cell carcinoma may come from hair follicles and could help the cancer grow.

A certain type of skin cell, marked by EGFR, produces a lot of IGF1 and helps hair follicles grow back faster.

The research found that a protein called PPARg is important for the formation and healing of sebaceous glands, which can regenerate independently from hair follicles.

Patients with SLE experienced non-scarring hair loss with fewer hair follicles, and DIF did not help identify lupus.

Patients with systemic lupus erythematosus and hair loss had fewer hair follicles and non-scarring alopecia; direct immunofluorescence was not helpful in diagnosing lupus in these cases.

Researchers found a new gene, hacl-1, that is active in mouse hair follicles during hair growth and may be important for hair biology.

The study found that hair follicles are above muscle connections in the scalp, which may help protect stem cell areas.

Each group of hair follicles on the scalp shares one muscle that helps control hair movement.

Overactivating Hedgehog signaling makes hair follicle cells in mice grow hair faster and create more follicles.

Stem cells rearrangement regenerates functional hair follicles, potentially treating hair loss.

Women with lupus experienced non-scarring hair loss with fewer hair follicles, and the test for lupus in hair was not helpful.

The scalp has a natural speckled pattern of increased pigment around hair follicles, possibly linked to local hormone production.

Different types of stem cells exist within individual skin layers, and they can adapt to damage, transplantation, or tumor growth. These cells are regulated by their environment and genetic factors. Tumor growth is driven by expanding, genetically altered cells, not long-lived mutant stem cells. There's evidence of cancer stem cells in skin tumors. Other cells, bacteria, and genetic factors help maintain balance and contribute to disease progression. A method for growing mini organs from single cells has been developed.

A genetic change in the EDAR gene causes the unique hair traits found in East Asians.

Different cell types work together to repair skin, and targeting them may improve healing and reduce scarring.

The skin's dermal layer contains true stem cells with diverse functions and interactions that need more research to fully understand.

The machine learning model effectively assesses the severity of hair loss and could help dermatologists with treatment decisions.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Sweat gland development involves two unique skin cell programs and a temporary skin environment.

Trichoscopy helps diagnose hairline recession causes in Egyptian women, with androgenetic alopecia being the most common.

Dermoscopy helps diagnose syphilitic alopecia, and treatment with penicillin can regrow hair.

Adult vibrissa follicle stem cells can regenerate hair follicles, glands, and skin.

The PTH-CBD injection improved hair regrowth better than the daily ruxolitinib pill in mice.

A PTH-based treatment improved hair regrowth better than ruxolitinib in a mouse model of hair loss.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

New treatment combining PRP and SVF increases hair density in 6-12 weeks for androgenetic alopecia patients.

Human scalp hair on mice can effectively monitor mercury exposure.

Hair color is not a risk factor for developing alopecia areata.