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Research 91–120 of 222
- Embryologic layers in dermatology: Developmental checkpoint disorders, diagnostic insight, and regenerative futures
- Retinoids in scarless skin regeneration: from molecular mechanisms to therapeutic strategies
- ラット精巣におけるビタミンD 代謝関連遺伝子の発現解析
- Facial hypertrichosis after isotretinoin therapy: Is it a side effect or coincidence?
- Generation of Genetically Modified Rats Using CRISPR/Cas9 Genome-Editing System to Reveal Novel Vitamin D Actions
- Dermal EZH2 simultaneously orchestrates dermal differentiation and epidermal proliferation during murine skin development
- Functional mapping of the mouse <i>hairless</i> gene promoter region
- Medications
- Acitretin for the Treatment of Psoriasis: A Case Report of Long-Lasting Alopecia
- Abstracts Pt.30
- An Automated Approach Towards Digital Photo-Trichogram for Hair Fall Diagnosis
- The epidermis: rising to the surface
- The role of PPAR<i>γ</i>-mediated signalling in skin biology and pathology: new targets and opportunities for clinical dermatology
- Topical Retinoids: Therapeutic Mechanisms in the Treatment of Photodamaged Skin
- A unique insertion/duplication in the VDR gene that truncates the VDR causing hereditary 1,25-dihydroxyvitamin D-resistant rickets without alopecia
- A Comprehensive Review of Acne Treatments: Unpacking the Chemical Structures and Effective Bioactive Compounds
- Gene therapy for alopecia in type II rickets model rats using vitamin D receptor-expressing adenovirus vector
- Physical and Functional Interaction between the Vitamin D Receptor and Hairless Corepressor, Two Proteins Required for Hair Cycling
- Behavioural characterization of Vitamin D receptor knockout mice
- Inactivation of the Vitamin D Receptor Enhances Susceptibility of Murine Skin to UV-Induced Tumorigenesis
- Dynamic regulation of retinoic acid-binding proteins in developing, adult and neoplastic skin reveals roles for β-catenin and Notch signalling
- The RAIG Family Member, GPRC5D, Is Associated with Hard-Keratinized Structures
- Hereditary 1,25-dihydroxyvitamin D-resistant rickets with alopecia resulting from a novel missense mutation in the DNA-binding domain of the vitamin D receptor
- Two Opposing Faces of Retinoic Acid: Induction of Stemness or Induction of Differentiation Depending on Cell-Type
- The vitamin D receptor functions as a transcription regulator in the absence of 1,25-dihydroxyvitamin D3
- Mechanisms of Action of Dehydroepiandrosterone
- A Humanized Mouse Model of Hereditary 1,25-Dihydroxyvitamin D–Resistant Rickets Without Alopecia
- PPAR‐γ signalling as a key mediator of human hair follicle physiology and pathology
- Mutations in the vitamin D receptor gene in four patients with hereditary 1,25-dihydroxyvitamin D-resistant rickets
- IL-1 Receptor–Knockout Mice Develop Epidermal Cysts and Show an Altered Innate Immune Response after Exposure to UVB Radiation