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Skin side effects from CDK4/6 inhibitors in breast cancer patients are generally mild and treatable, allowing most patients to continue treatment.

There is no cure for alopecia areata, but treatments like JAK inhibitors show promise.

Treating alopecia areata is difficult due to limited approved treatments, but new therapies like JAK inhibitors show promise.

The document concludes that managing side effects of MS therapies is crucial for treatment success and patient adherence.

B cells can both help and hinder the body's defense against melanoma.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Manipulating cell cleanup processes could help treat hair loss.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

Canine pemphigus foliaceus involves significant immune activity and shares similarities with human pemphigus.

Hair growth phase and certain genes can speed up wound healing, while an inflammatory mediator can slow down new hair growth after a wound. Understanding these factors can improve tissue regeneration during wound healing.

Unconventional lymphocytes are important for quick immune responses and healing of skin and mucosal barriers.

T-cell reconstitution after thymus transplantation can cause hair whitening and loss.

Leptin-deficient mice, used as a model for Type 2 Diabetes, have delayed wound healing due to impaired contraction and other dysfunctional cellular responses.

Alopecia Areata is an autoimmune condition causing hair loss with no cure and treatments that often don't work well.

Controlled delivery of specific RNA and IL-4 restored hair growth in mice with autoimmune alopecia.

FOXN1 gene variants cause low T cells and immune issues from birth.

Psoriasis is linked to other autoimmune diseases and involves a specific inflammatory process.

Leflunomide is more likely to help treat alopecia areata than cause it.

Dandruff is linked to increased T cells and weakened immune protection in hair follicles.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

ATIR101 improves survival in stem cell transplant patients; Australian stem cell treatment decisions are influenced by regulation changes.

Deleting the BRD4 protein in certain skin cells causes hair loss and skin inflammation.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

Gamma delta T cells in the skin help with healing and defense but can also cause autoimmune issues, and more research is needed to understand how they are activated.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

taVNS reduces vitiligo symptoms in mice.

New treatments for immune disorders caused by FOXN1 deficiency are promising.

MAD2B slows down the growth of skin cells that are important for hair development by interacting with TCF4.

A specific DNA sequence caused hair loss in male mice by activating immune cells and increasing a certain immune signal.

New effective scar treatments are urgently needed due to the current options' limited success.