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PDRN, derived from salmon sperm, shows promise in healing wounds, reducing inflammation, and regenerating tissues, but more research is needed to understand its mechanisms and improve its use.

Some growth factors, while important for normal body functions, can cause diseases when not regulated properly.

Radiation increases cell death in hair follicles, but certain treatments can protect against this effect.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

Hair follicles repair 3D injuries using a 2D healing process.

Different types of sun exposure can damage skin cells and affect healing, with chronic exposure being more harmful, and certain immune cells help in the repair process.

Melatonin protects skin against UV damage by regulating various cellular processes.

Radiation damages salivary glands by harming blood vessels, but antioxidants might help protect them.

Stress activates a special function of the Vitamin D receptor with the help of c-Jun, which can also prevent cell death.

Cepharanthine, a Japanese hair loss drug, shows promise as a COVID-19 treatment but needs more testing.

FGF18 treatment during hair's resting phase can protect against hair loss from radiation.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Human foreskin does not show aging or reduced cell growth after radiation, and H2A.J is not a good marker for radiation-induced aging.

Hair greying is caused by oxidative stress damaging hair follicles and melanocytes.

Arctiin helps protect hair cells from damage and death caused by oxidative stress.

The document concludes that understanding adult stem cells and their environments can help improve skin regeneration in the future.

Using A132 hair dye with sunlight can cause skin damage and hair loss.

Melatonin may protect inner ear cells from damage by reducing cell death and oxidative stress, potentially treating sudden hearing loss.

Nitroxide drugs can safely and effectively treat age-related diseases like macular degeneration and cardiovascular issues.

Cancer treatments often cause hair disorders, significantly affecting patients' quality of life, and better management methods are needed.

N-acetylcysteine may prevent hair loss caused by chemotherapy.

Vesicles from irradiated mouse cheek skin help cells survive radiation.

Cell death shapes skin stem cell environments, affecting inflammation, repair, and cancer.

Caspases are enzymes important for both cell death and various non-lethal cell functions, affecting head development and hair growth, with different caspases playing specific roles.

UV radiation can help sterilize wounds and promote healing but requires careful use to avoid damaging cells.

Agaricus bisporus derived β-Glucan could be an effective cervical cancer treatment with antimicrobial and antioxidant properties.

Regulating cell death in hair follicles can help prevent hair loss and promote hair growth.

Chemotherapy causes hair loss by damaging hair follicles and stem cells, with more research needed for prevention and treatment.

ATP increases melanin production in skin after UV exposure, with the P2X7 receptor being crucial for this process.

Hair grows faster in the morning and is more vulnerable to damage from radiation due to the internal clock in hair follicle cells.