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The 2015 Hair Research Congress concluded that stem cells, maraviroc, and simvastatin could potentially treat Alopecia Areata, topical minoxidil, finasteride, and steroids could treat Frontal Fibrosing Alopecia, and PTGDR2 antagonists could also treat alopecia. They also found that low-level light therapy could help with hair loss, a robotic device could assist in hair extraction, and nutrition could aid hair growth. They suggested that Alopecia Areata is an inflammatory disorder, not a single disease, indicating a need for personalized treatments.

Blocking Prostaglandin D₂ may help treat hair loss.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Lithocholic acid helps hair growth and regeneration in alopecia by activating vitamin D receptors.

Gypenosides from Gynostemma pentaphyllum were found to have anti-aging effects, increasing skin collagen and reducing wrinkles.

New treatments for hair loss may target specific pathways and generate new hair follicles.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

Dexamethasone affects hair growth by altering levels of proteins that either promote or inhibit hair follicle growth.

PGD2 stops hair growth and is higher in bald men with AGA.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

Hair loss in Androgenetic alopecia (AGA) is due to altered cell sensitivity to hormones, not increased hormone levels. Hair growth periods shorten over time, causing hair to become thinner and shorter. This is linked to miscommunication between cell pathways in hair follicles. There's also a change in gene expression related to blood vessels and cell growth in balding hair follicles. The exact molecular causes of AGA are still unclear.

Targeting the PGD2-DP2 pathway may help treat hair loss.

PGD2 increases androgen receptor activity in hair cells, which could be targeted to treat hair loss.

Wnt3a activates certain genes in hair follicle cells, including a newly discovered one, EP2, which may affect hair growth.

Topical cetirizine improves hair density and thickness in androgenetic alopecia, but more research is needed.

SFRP2 and PTGDS may be key factors in female hair loss.

Genetic factors could lead to personalized treatments for hair loss.

The PTGER2 gene is highly active in Cashmere goat skin and its activity changes with the hair growth cycle.

Prostaglandin D₂ might be targeted for new male pattern baldness treatments.

Fat cells in the skin help start healing and form important repair cells after injury.

Minoxidil helps hair growth by activating the β-catenin pathway.

Multiple treatments work best for hair loss.

Skin and hair can regenerate after injury due to changes in gene activity, with potential links to how cancer spreads. Future research should focus on how new hair follicles form and the processes that trigger their creation.

BMP4-related anomalies can cause a wide range of eye, brain, and hand/foot problems, and new cases show this variability.

Uterine leiomyomas don't significantly change gene expression in the scalp of people with Central Centrifugal Cicatricial Alopecia.

Treating hair loss effectively may require a multi-sided approach, using different treatments together, and topical treatments could be more effective and safer than oral ones.

Multi-omics techniques help understand the molecular causes of androgenetic alopecia.