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- 973 Dipeptidyl-peptidase 4 (DPP4)-positive fibroblasts are responsible for secretion of pro-fibrotic matrix proteins in the human skin
- Identification of immune microenvironment changes, immune-related pathways and genes in male androgenetic alopecia
- Langerin+ Dendritic Cells in Cutaneous Fibrosis: The TGF-β1 Signaling Axis
- Effects of ginsenoside Rb1 on hypertrophic scar remodeling in rabbit model
- Integrin β1 Establishes Liver Microstructure and Modulates Transforming Growth Factor β during Liver Development and Regeneration
- Axolotl-derived decellularized skin ECM as a pro-regenerative scaffold for attenuating fibrotic wound healing
- 1325 Age-dependent loss of the stemness and antimicrobial defense function of dermal fibroblasts is mediated by TGFbeta
- Fibroblast Lineage Switching as the Developmental Origin of Scarring and Target for Regenerating Healing
- Toward understanding scarless skin wound healing and pathological scarring
- ADM Scaffolds Generate a Pro-regenerative Microenvironment During Full-Thickness Cutaneous Wound Healing Through M2 Macrophage Polarization via Lamtor1
- Recombinant Human Annexin A5 Ameliorates Localized Scleroderma by Inhibiting the Activation of Fibroblasts and Macrophages
- The Secretome of Irradiated Peripheral Mononuclear Cells Attenuates Hypertrophic Skin Scarring
- Intradermal adipocytes mediate fibroblast recruitment during skin wound healing
- Enter the Matrix: Fibroblast-immune cell interactions shape extracellular matrix deposition in health and disease.
- Divergent molecular signatures of regeneration and fibrosis during wound repair
- Interleukin‐10 Derived Apoptotic Vesicles Enhance Scarless Skin Healing by Modulating Fibroblast Metabolism and Fibrosis Pathways
- Antiviral drugs prolong survival in murine recessive dystrophic epidermolysis bullosa
- Harnessing the Biomimetic Effect of Macromolecular Crowding in the Cell-Derived Model of Clubfoot Fibrosis
- An ERK-dependent molecular switch antagonizes fibrosis and promotes regeneration in spiny mice ( <i>Acomys</i> )
- Helminth protein enhances wound healing by inhibiting fibrosis and promoting tissue regeneration
- The senescence-like activity of BMS-470539 is associated with anti-fibrotic actions in models of dermal fibrosis
- Profibrotic Subsets of SPP1+ Macrophages and POSTN+ Fibroblasts Contribute to Fibrotic Scarring in Acne Keloidalis
- Deletion of adipocyte Sine Oculis Homeobox Homolog 1 prevents lipolysis and attenuates skin fibrosis
- Fibromodulin reduces scar formation in adult cutaneous wounds by eliciting a fetal-like phenotype
- Profibrotic Molecules Are Reduced in CRISPR-Edited Emery–Dreifuss Muscular Dystrophy Fibroblasts
- Electrospun Nanofibers for Improved Angiogenesis: Promises for Tissue Engineering Applications
- Co-opting Developmental Signaling Pathways to Promote Wound Healing
- Mesenchymal Stem Cells from Adipose Tissue in Clinical Applications for Dermatological Indications and Skin Aging
- Retinoids in scarless skin regeneration: from molecular mechanisms to therapeutic strategies
- Fibroproliferative genes are preferentially expressed in central centrifugal cicatricial alopecia