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- Lipoatrophy and severe metabolic disturbance in mice with fat-specific deletion of PPARγ
- Maternal PPARγ protects nursing neonates by suppressing the production of inflammatory milk
- The role of PPAR<i>γ</i>-mediated signalling in skin biology and pathology: new targets and opportunities for clinical dermatology
- Peroxisome proliferator-activated receptors (PPARs) and PPAR agonists: the ‘future’ in dermatology therapeutics?
- Expression of Peroxisome Proliferator Activated Receptors (PPARs) in Human Hair Follicles and PPARα Involvement in Hair Growth
- Functions of peroxisome proliferator‐activated receptors (PPAR) in skin homeostasis
- PPAR-<i>γ</i> Agonists and Their Role in Primary Cicatricial Alopecia
- Advanced Inhibition of Undesired Human Hair Growth by PPARγ Modulation?
- PPAR‐γ signalling as a key mediator of human hair follicle physiology and pathology
- Absence of vitamin D receptor (VDR)‐mediated PPARγ suppression causes alopecia in VDR‐null mice
- Epidermal PPARγ Is a Key Homeostatic Regulator of Cutaneous Inflammation and Barrier Function in Mouse Skin
- Progressive expression of PPARGC1α is associated with hair miniaturization in androgenetic alopecia
- Non-clinical safety evaluation and risk assessment to human of aleglitazar, a dual PPAR α/γ agonist, and its major human metabolite
- Mechanism of PPARα agonist in alopecia areata
- Keratin 79 is a PPARA target that is highly expressed by liver damage
- Astilbin Activates the Reactive Oxidative Species/PPARγ Pathway to Suppress Effector CD4+ T Cell Activities via Direct Binding With Cytochrome P450 1B1
- FAM19A5 negatively regulates cutaneous wound healing via PPARD-dependent inhibition of keratinocyte migration
- The Role of PPARβ/δ-Related Lipid Metabolism in High-Altitude Adaptation of Yak Coat Based on Proteomics and Metabolomics
- Integrative Proteo‐Transcriptomic Characterization of Androgenetic Alopecia Identifying ME1‐Mediated PPAR Signaling as a Potential Mediator
- In vitro hair growth-promoting effects of araliadiol via the p38/PPAR-γ signaling pathway in human hair follicle stem cells and dermal papilla cells
- Early skin seeding regulatory T cells modulate PPARγ-dependent skin pigmentation
- Long non-coding RNA AL136131.3 inhibits hair growth through mediating PPARγ in androgenetic alopecia
- Loss of adipocyte phospholipase gene PLAAT3 causes lipodystrophy and insulin resistance due to inactivated arachidonic acid-mediated PPARγ signaling
- 599 PPARg signaling modulation protects from hair follicle bulge stem cell damage and cyclophosphamide-induced hair follicle cytotoxicity
- 1380 PPAR-γ modulation as a novel therapeutic strategy for scarring alopecia
- 269 The PPAR-γ modulator, N-acetyl-GED, protects from epithelial-mesenchymal-transition to human hair follicles: Potential relevance for scarring alopecia
- Author response: Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ
- Decision letter: Loss of Dnmt3a and Dnmt3b does not affect epidermal homeostasis but promotes squamous transformation through PPAR-γ
- Faculty Opinions recommendation of Hair follicle stem cell-specific PPARgamma deletion causes scarring alopecia.
- Pathogenicity of<i>Ureaplasma urealyticum</i>and<i>Ureaplasma parvum</i>in the lower genital tract of female BALB/c mice