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The article concludes that hair loss is a common side effect of drugs treating skin cancer by blocking the hedgehog pathway, but treatment should continue, and more selective drugs might prevent this side effect.

Monkeys with hair loss during pregnancy showed higher stress hormone levels and invested differently in their offspring.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Skin patterns are formed by simple reaction-diffusion mechanisms.

Light can turn on hair growth cells through a nerve path starting in the eyes.

PTEN helps control the number and health of skin stem cells by working with the protein BMAL1.

The best animal model for studying male-pattern baldness is the stumptailed macaque, not rats or mice.

Circadian clock genes are important for hair growth and may affect aging-related hair loss and graying.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Hair can regrow in adult mice's skin after injury, and this process can be boosted by increasing Wnt7a, a protein. This could potentially help treat baldness and change our understanding of hair growth.

Mice with too much sPLA₂-IIA have hair loss and poor wound healing due to abnormal hair growth and stem cell depletion.

A gene mutation causes early keratinocyte maturation leading to hair loss in Olmsted syndrome.

Stress worsens hair loss in androgenetic alopecia.

The rhg mutation in mice affects the Oat gene, causing hair growth issues and other symptoms.

miR-195-5p reduces hair growth ability in cells by blocking a specific growth signal.

Gamma rays did not change hair follicle density but increased white and hypopigmented hairs in mice.

New hair can grow from large wounds in mice, but less so as they age, involving reprogramming of skin cells and specific molecular pathways.

Gene therapy, especially using atoh1, shows promise for creating functional sensory hair cells in the inner ear, but dosing and side effects need to be managed for clinical application.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Sonic hedgehog signaling is crucial for hair growth and maintaining hair follicle identity.

Biofield Energy Healing may increase hair growth by enhancing cell proliferation.

Biofield Energy Healing significantly increased hair growth by making hair root cells grow more.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Vitamin D and its receptor regulate skin functions like cell growth, immunity, hair cycle, and tumor prevention.

Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

Skin cells produce and activate vitamin D, which regulates skin functions and supports hair growth.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.

New research shows that skin diversity is influenced by different types of dermal fibroblasts and their development, especially involving the Wnt/β-catenin pathway.

Certain signals and genes play a key role in hair growth and regeneration, and understanding these could lead to new treatments for skin regeneration.