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Research 22
- PBX homeobox 1 enhances hair follicle mesenchymal stem cell proliferation and reprogramming through activation of the AKT/glycogen synthase kinase signaling pathway and suppression of apoptosis
- <i>Hoxc13</i> mutant mice lack external hair
- Hoxc13 polyglycine repeat gain-of-function drives mammalian integument evolution by altering targeted genes and interactions
- Signaling pathways in hair aging
- Meis1 Regulates Epidermal Stem Cells and Is Required for Skin Tumorigenesis
- The inconsistent regulation of HOXC13 on different keratins and the regulation mechanism on HOXC13 in cashmere goat (Capra hircus)
- HOXC13 Is Involved in the Regulation of Human Hair Keratin Gene Expression
- PBX1-SIRT1 Positive Feedback Loop Attenuates ROS-Mediated HF-MSC Senescence and Apoptosis
- The HOXC13-controlled expression of early hair keratin genes in the human hair follicle does not involve TALE proteins MEIS and PREP as cofactors
- PBX1 Attenuates Hair Follicle-Derived Mesenchymal Stem Cell Senescence and Apoptosis by Alleviating Reactive Oxygen Species-Mediated DNA Damage Instead of Enhancing DNA Damage Repair
- Hox in hair growth and development
- Hox genes in development and beyond
- Water extract of cacumen platycladi promotes hair growth through the Akt/GSK3β/β-catenin signaling pathway
- MSCs and their exosomes: a rapidly evolving approach in the context of cutaneous wounds therapy
- Developmentally regulated expression of integrin alpha-6 distinguishes neural crest derivatives in the skin
- Smurf2-induced degradation of SMAD2 causes inhibition of hair follicle stem cell differentiation
- Status of research on the development and regeneration of hair follicles
- Brg1 Governs a Positive Feedback Circuit in the Hair Follicle for Tissue Regeneration and Repair
- Maryland Community Colleges Technology Needs Assessment Survey. Final Technical Report.
- Targeting integrin pathways: mechanisms and advances in therapy
- Cloning of human, murine, and marsupial keratin 7 and a survey of K7 expression in the mouse
- Weathering the Storm: Harnessing the Resolution of Inflammation to Limit COVID-19 Pathogenesis