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The gene p53 is crucial for removing damaged cells to allow for healthy tissue renewal.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.

CXXC5 is a protein that controls cell growth and healing processes, and changes in its activity can lead to diseases like cancer and hair loss.

The p53 protein has complex, sometimes contradictory functions, including tumor suppression and promoting cell survival.

Ginsenoside Rg1 protects mouse skin from UVB damage and helps control inflammation.

Short telomeres contribute to aging and cancer, and while telomerase can delay aging, it may also promote cancer.

Histone demethylases play a key role in the development of many diseases and may be targets for treatment.

Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Mice can regenerate ear tissue without the p53 protein.

The Wnt signaling pathway is not a major factor in the development of keratoacanthoma, a type of skin tumor.

Short-term finasteride treatment may lower some cell death factors in prostate cancer cells.

The S100A4 protein is more common in psoriatic skin and could be a target for treating psoriasis.

Photodynamic therapy is the preferred treatment for skin precancer due to its effectiveness and safety.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

Acne treatment with isotretinoin increases the presence of p53, a protein, in skin and oil glands, which may help reduce acne severity.

Understanding how cells die in the skin is important for treating skin diseases and preventing hair loss.

Maintaining DNA health in stem cells is key to preventing aging and tissue breakdown.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Blocking both main energy pathways can stop hair follicle stem cell-induced skin cancer growth.

Distinct β-catenin patterns are linked to cell growth, not cell death, in lung cancer.

Cellular senescence has both cancer-blocking and cancer-promoting effects, and targeting senescent cells may improve health and lifespan.

Stem cell niches are crucial for regulating stem cell behavior and tissue health, and their decline can impact aging and cancer.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Most adrenal cortex tumors are benign and non-secreting, but proper diagnosis and treatment are important due to the rare possibility of cancer.

Gene therapy shows promise for treating skin disorders and cancer, but faces technical challenges.

N-acetylcysteine may prevent hair loss caused by chemotherapy.

UV radiation causes skin cancer, but sunscreens and certain drugs can help prevent it.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.