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Research 31–60 of 476
- Alopecia Areata
- Alopecia areata: Animal models illuminate autoimmune pathogenesis and novel immunotherapeutic strategies
- The Role of Hair Follicle Immune Privilege Collapse in Alopecia Areata: Status and Perspectives
- Mathematical model for alopecia areata
- Advance Understanding and New Treatment of Alopecia Areata
- DISRUPTION OF HAIR FOLLICLE IMMUNE PRIVILEGE IN ALOPECIA AREATA: ENIGMATIC MECHANISMS AND EMERGING CONCEPTS
- Editors’ Picks
- Author response: Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- 331 Mitochondrial stress contributes to atopic dermatitis
- Decision letter: Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- 원형탈모증(alopecia areata)의 최신 이해와 치료
- 328 The transcription factor CEBPB is a novel hub gene and multi-functional disease driver in Psoriatic skin inflammation
- 516 Possible role of ILC1 in the pathogenesis of alopecia areata (AA)
- Janus Kinase Inhibitors against Other Biological Treatments in Alopecia Areata
- ULBP3: a marker for alopecia areata incognita
- Inhibiting Janus kinases to treat alopecia areata
- Innate lymphoid cells type 1 may be new, non-antigen-specific player in the pathogenesis of alopecia areata
- Epigallocatechin-3 Gallate Inhibits STAT-1/JAK2/IRF-1/HLA-DR/HLA-B and Reduces CD8 MKG2D Lymphocytes of Alopecia Areata Patients
- Alopecia areata is characterized by dysregulation in systemic type 17 and type 2 cytokines, which may contribute to disease‐associated psychological morbidity
- Butyrophilin-like 2 (BTNL2) to the Rescue: controlling autoaggressive dermal gamma delta (GD) T lymphocytes in human hair follicles (HFs) 3747
- 431 Innate lymphoid cells type 1 may be new, non-antigen-specific player in the pathogenesis of alopecia areata
- Genome-wide association study in alopecia areata implicates both innate and adaptive immunity
- Involvement of ILC1-like innate lymphocytes in human autoimmunity, lessons from alopecia areata
- Increased blood levels of NKG2D+CD4+ T cells in patients with alopecia areata
- High-throughput T cell receptor sequencing identifies clonally expanded CD8+ T cell populations in alopecia areata
- High throughput T cell receptor sequencing identifies clonally expanded CD8+ T cell populations in Alopecia Areata
- Single-cell analysis of temporal immune cell dynamics in alopecia areata reveals a causal role for clonally expanded CD8+ T cells in disease
- SOCS3 treatment prevents the development of alopecia areata by inhibiting CD8+ T cell-mediated autoimmune destruction
- Th1 effector CD4 T cells rely on IFN-γ production to induce alopecia areata
- Possible Involvement of Skin‐Resident Memory T Cells in Refractory Chronic Alopecia Areata