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Hair follicle development in cashmere goats involves dynamic changes in proteins and metabolites, with key roles for oxytocin, MAPK, and Ca2+ pathways.

Neuroactive steroids and the enzyme 5α-reductase might be involved in the development of Tourette's syndrome.

Exosomes from stem cells help hair regrowth by activating a specific signaling pathway.

Melatonin affects certain genes and pathways involved in cashmere goat hair growth.

PFS and PSSD are similar conditions with persistent sexual dysfunction after stopping medication.

Melatonin can increase cashmere yield by altering gene expression and restarting the growth cycle early.

Deleting the EDAR gene in Cashmere goats affects genes and proteins related to hair growth.

A gene mutation in Lama3 is linked to a common type of hair loss.

Neuroactive steroids could become safe, effective treatments with more understanding of their complex brain actions and metabolism.

New targets for making and using brain-synthesized steroids could lead to better treatments for brain disorders and alcoholism.

Neuroactive steroids may affect the risk and treatment of alcohol use disorders.

Finasteride treats enlarged prostate and hair loss, but may cause side effects in some patients.

Different amounts of daylight affect cashmere growth in goats by changing the activity of certain genes and molecules.

Lower levels of certain brain chemicals are linked to worse PTSD symptoms in men.

Cinnamic acid may help hair grow by activating oxytocin receptors.

Some people experience lasting sexual, psychological, and sleep problems after using finasteride or SSRI antidepressants, possibly due to similar underlying causes.

Gut microbes significantly affect brain steroid levels.

Gut flora changes could potentially indicate depression, but more research is needed.

Finasteride treatment changes brain steroid levels and receptors, affecting brain function even after stopping treatment.

Certain steroids in the brain affect mood and symptoms of depression, and treatments targeting these steroids show promise for improving these symptoms.

Finasteride use changes brain chemicals, causing lasting sexual issues and anxiety/depression.

Post-finasteride patients show changed neuroactive steroid levels, possibly causing erectile dysfunction and depression.

Neuroactive steroids show promise for treating mental and neurological disorders by targeting GABA_A receptors.

Some hormone-related drugs may protect brain cells in Parkinson's disease differently in men and women.

The enzymes 5α-reductase and 3α/β-hydroxysteroid oxidoreductase help create brain-active substances from progesterone and testosterone, which could be used for treatment, but more research is needed to ensure their safety and effectiveness.

Lipids may help treat hair loss by promoting hair growth through the HIF-1 pathway.

Finasteride and dutasteride reduce unwanted movements from Parkinson's disease treatment by normalizing certain brain signals.

Low dose finasteride decreases certain steroids, possibly increasing depression risk.

Early changes in brain chemicals affect how a drug reduces alcohol intake in rats.

Myristoleic acid helps hair growth by boosting cell growth and recycling processes in hair follicle cells.