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Research 61–90 of 1000+
- Regulatory T cells in skin
- Regulatory T-Cells: Potential Regulator of Tissue Repair and Regeneration
- KLK5 and KLK7 Ablation Fully Rescues Lethality of Netherton Syndrome-Like Phenotype
- Neurosteroids, neuroactive steroids, and symptoms of affective disorders
- The concealed side of caspases: beyond a killer of cells
- Plakophilin 1 suppresses keratinocyte innate immune responses through DExD/H helicases
- Mechanisms regulating skin immunity and inflammation
- Extraskeletal actions of vitamin D
- Beyond Expectations
- Management of congenital ichthyoses: European guidelines of care, part two
- Lineage Tracing of Hair Follicle Stem Cells in Epidermal Whole Mounts
- HuR ablation destabilizes Foxp3 mRNA and impairs regulatory T cell function, contributing to an autoimmune phenotype
- CD4 expression controls epidermal stem cell balance
- Therapeutic potential and pharmacological insights of total glucosides of paeony in dermatologic diseases: a comprehensive review
- Anaesthetics and platelets.
- Mast-cell driven dermal white adipose tissue remodeling amplifies inflammation and fibrosis in atopic dermatitis
- Epidermal Notch1 recruits innate lymphoid cells to orchestrate normal skin repair
- How to diagnose a lipodystrophy syndrome
- RhoA promotes epidermal stem cell proliferation via PKN1-cyclin D1 signaling
- Geriatric Dermatologic Therapy
- Notch Signaling Regulates Late-Stage Epidermal Differentiation and Maintains Postnatal Hair Cycle Homeostasis
- Modulation of the differentiated phenotype of keratinocytes of the hair follicle and from epidermis
- Epidermal keratinocytes initiate wound healing and pro-inflammatory immune responses following percutaneous schistosome infection
- Tissue Resident Foxp3+ Regulatory T Cells: Sentinels and Saboteurs in Health and Disease
- ILC3s as central regulators of mucosal homeostasis and disease pathogenesis (Review)
- Regulatory T cells in dominant immunologic tolerance
- Advances in single-cell transcriptomics in animal research
- Keratins as an Inflammation Trigger Point in Epidermolysis Bullosa Simplex
- A Spontaneous Deletion within the Desmoglein 3 Extracellular Domain of Mice Results in Hypomorphic Protein Expression, Immunodeficiency, and a Wasting Disease Phenotype
- Hair follicles modulate skin barrier function