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Research 391–420 of 1000+
- Friends Turned Foes: Angiogenic Growth Factors beyond Angiogenesis
- Mapping of cis-acting expression quantitative trait loci in human scalp hair follicles
- The role of protein lactylation in skin diseases: from molecular mechanisms to potential therapeutics
- Plain language summaries
- Author response: The molecular basis for ANE syndrome revealed by the large ribosomal subunit processome interactome
- Decision letter: The molecular basis for ANE syndrome revealed by the large ribosomal subunit processome interactome
- Matriptase-2, A Novel Suppressor of Hepcidin.
- A polygenic mouse model of psoriasiform skin disease in CD18-deficient mice.
- Activated Kras Alters Epidermal Homeostasis of Mouse Skin, Resulting in Redundant Skin and Defective Hair Cycling
- <i>BUZZ</i>: an essential gene for postinitiation root hair growth and a mediator of root architecture in <i>Brachypodium distachyon</i>
- Decision letter: Plant Trans-Golgi Network/Early Endosome pH regulation requires Cation Chloride Cotransporter (CCC1)
- The hairless (hr) gene is involved in the congential hypotrichosis of Valle del Belice sheep
- Current Genetics in Hair Diseases
- Cell death by cornification
- Alopecia in a Viable Phospholipase C Delta 1 and Phospholipase C Delta 3 Double Mutant
- Gray Hair: From Preventive to Treatment
- 545 Skin Organoids derived from NCSTN mutated patient-induced pluripotent stem cells recapitulate Hidradenitis Suppurativa pathogenic hallmarks
- Aging of hair
- Aging in the sebaceous gland
- Deletion of an enhancer in FGF5 is associated with ectopic expression in goat hair follicles and the cashmere growth phenotype
- FGF5 is a crucial regulator of hair length in humans
- Towards Defining the Pathogenesis of the Hairless Phenotype
- An essential role for <i>Rxrα</i> in the development of Th2 responses
- Heterogeneity in the genetic alterations and in the clinical presentation of acrodermatitis enteropathic: Case report and review of the literature
- RISK FACTORS, PREVALENCE AND DIAGNOSIS OF HUTCHISON GILFORD SYNDROME WITH SPECIAL REFERENCE TO CASE REPORTS
- 547 CDK9 Kinase Activation in Association with AFF1-SEC Initiate Epidermal Progenitor differentiation
- Keratins as an Inflammation Trigger Point in Epidermolysis Bullosa Simplex
- Molecular Genetics of the PI3K-AKT-mTOR Pathway in Genodermatoses: Diagnostic Implications and Treatment Opportunities
- Pathological Mechanisms Involved in Epidermolysis Bullosa Simplex: Current Knowledge and Therapeutic Perspectives
- Protein degradation: expanding the toolbox to restrain cancer drug resistance