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The timing of when the gene Bmal1 is active affects aging and survival, with its absence during development, not adulthood, leading to premature aging.

Different body areas in mice produce different hair types due to interactions between skin layers.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Androgen receptors in the mouse brain may explain cognitive and mood changes in prostate cancer treatment.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

Mouse skin color ranges from pink to black, depending on their hair growth cycle.

Removing Dicer from pigment cells in newborn mice causes early hair graying and changes in cell migration molecules.

Meis2 is essential for touch sensation and nerve function in mice.

Follistatin and LIN28B together improve the ability of inner ear cells in mice to regenerate into hearing cells.

Activin A and follistatin control when ear hair cells form in mice.

Notch/CSL signaling controls hair follicle differentiation through Wnt5a and FoxN1.

The project created a standardized system for classifying skin lesions in lab rats and mice.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Autophagy is crucial for normal sebaceous gland function and sebum composition.

Altered retinoid metabolism in cicatricial alopecia suggests a balanced vitamin A diet may prevent the condition.

Activating β-catenin increases melanocytes and decreases Schwann cells.

Apoptosis, not oxidative stress, is linked to aging in mice with mtDNA mutations.

TRPA1 is crucial for mechanical sensitivity in skin sensory neurons.

The hedgehog signaling pathway is crucial for hair growth but not for the initial creation of hair follicles.

Jumonji genes are important for development and their mutations can cause abnormalities, especially in the heart and brain.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Fatty acid transport protein 4 is essential for skin and hair development.

Stump-tailed macaque best for researching hair loss causes and treatments.

Blocking Wnt/β-catenin signaling with EGF receptor is necessary for proper hair growth.

GasderminA3 is important for normal hair cycle transitions by controlling Wnt signaling.

Signals from skin cells controlled by Rac proteins help turn certain precursor cells into white fat cells.

Different types of inactive melanocyte stem cells exist with unique characteristics and potential to develop into other cells.

aPKCλ is essential for hair follicle stem cell maintenance and wound healing.

Skin needs dermal β-catenin activity for hair growth and skin cell multiplication.