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Damaging mitochondrial DNA in mice speeds up aging due to increased reactive oxygen species, not through the p53/p21 pathway.

Growing hair follicles have high mitochondrial activity and ROS in specific regions, aiding hair formation.

Tissue-specific variation in mutant load complicates genetic counseling and prenatal diagnosis.

Stopping mitochondrial respiration can prevent brain cancer spread in skin cancer patients, and plant compound β-sitosterol could help achieve this.

Minoxidil protects heart and improves recovery.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Serenoa repens extract helps regrow hair and repair hair loss by activating certain cell growth pathways.

Minoxidil prevents serotonin loss from MDMA by affecting potassium channels and increasing Akt phosphorylation.

Improving mitochondrial health may better treat atherosclerosis than antioxidants.

Smoking reduces antioxidants and causes DNA damage in hair follicles.

Using A132 hair dye with sunlight can cause skin damage and hair loss.

Mitochondrial therapy and platelet-rich plasma therapy both stimulated hair regrowth in aging mice, with mitochondrial therapy showing similar effectiveness to plasma therapy.

Environmental pollutants like glyphosate, fluoride, and electromagnetic fields can cause health problems by damaging mitochondria and affecting metabolism.

Minoxidil helps hair growth by promoting stem cell differentiation through increased ATP synthase activity.

A patient with a rare disease had a unique genetic mutation linked to their symptoms.

Hair loss in men might be linked to changes in cell energy factories.

Mitochondrial problems in diabetic nerve damage might cause pain by lowering the production of certain nerve-related steroids.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Skin cells from people with Epidermolysis Bullosa Simplex have abnormally placed and less active mitochondria.

Tetrathiomolybdate reduces hair growth marker in skin cells by boosting harmful oxygen molecules, but effects can be reversed.

MOF controls key genes for skin development by regulating mitochondrial and ciliary functions.

Diabetic neuropathy in mice is linked to poor mitochondria function and lower brain hormone production.

MFN2 mutations cause mitochondrial problems, leading to more upper body fat and lower leptin levels.

Mitochondrial dysfunction is linked to various skin conditions and could be a target for treatments.

Mutations in mitochondrial DNA might significantly contribute to the development of Polycystic Ovarian Syndrome.

Defective T cell metabolism can cause early skin aging and poor hair follicle stem cell function.

Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

A protein called MPZL3 in mitochondria slows down hair growth and could be a target for treating hair growth disorders.

Butyrate helps hair grow longer, improves hair cell health, increases pigment production, and boosts hair's natural defenses.

Substance P may contribute to hair loss by increasing oxidative stress and mitochondrial activity in hair follicles.