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Nanoparticles can deliver vaccines through hair follicles, triggering immune responses and providing protection.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

The study found that a specific genetic variation in the TNFα gene is significantly linked to Alopecia Areata in the Jordanian Arab population.

Mesenchymal stem cells show promise for treating various skin conditions and may help regenerate hair.

Intestinal intraepithelial lymphocytes are crucial for gut immunity and maintaining the mucosal barrier.

UVB exposure in human skin causes macrophages to produce more IL-10 and less IL-12, leading to immunosuppression.

Malt1 protease is essential for regulatory T cell function and could be targeted to boost antitumor immunity.

PPAR-γ is important for healthy hair and its problems, and more research on PPAR-γ treatments is needed.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

A new hydrogel with stem cells from the human umbilical cord speeds up healing in diabetic wounds.

The study concludes that regulating apoptosis could lead to new treatments for various skin and hair conditions.

JAK inhibitors show promise for treating various skin diseases.

Immune activities and specific genes are important in male pattern baldness.

Innate lymphoid cells help control skin bacteria by regulating sebaceous glands.

Alopecia areata is caused by immune system attacks on hair follicles, often triggered by viral infections.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Alopecia areata can be triggered by specific immune cells without genetic or environmental factors.

Contact sensitizers like DCP and SADBE are the most effective treatments for alopecia areata.

JAK inhibitors show promise in effectively treating hair loss from alopecia areata.

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Volatile organic compounds can cause inflammation and increase the risk of autoimmune diseases.

HLA-DRB5 and other genes may be linked to alopecia universalis.

Antigen presenting cells around hair follicles are crucial in SLE-related hair loss.

Autophagy helps keep skin healthy and may improve treatments for skin diseases.

Mast cells and CD8 T cells interact closely in skin diseases, affecting each other's behavior and contributing to conditions like psoriasis and eczema.

Our microbiome may affect the development of the hair loss condition Alopecia Areata, but more research is needed to understand this relationship.

B2m-free HLA variants may be a new class of HLA important in immune responses and diseases.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

Dandruff might be caused by changes in how hair follicles naturally release oils and an immune response to this imbalance.

The secretome from mesenchymal stromal cells shows promise for improving facial nerve injury treatment.