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Tribuloside can increase skin pigmentation by enhancing melanin production and distribution.

Different types of inactive melanocyte stem cells exist with unique characteristics and potential to develop into other cells.

The human scalp has different types of pigment cells in hair follicles with varying abilities to produce pigment.

Lenalidomide helps hair follicle stem cells turn into melanocytes, which may improve repigmentation in vitiligo.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

Stress causes hair graying by overactivating nerves that deplete color-giving stem cells.

Hair follicle stem cells prevent melanocyte stem cells from differentiating by controlling retinoic acid levels.

Melanocyte transplantation can safely restore skin color, especially in stable vitiligo, but must be chosen carefully based on the disease phase.

Hair graying is caused by the loss of pigment cells due to poor maintenance of stem cells in the hair follicle.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

Injecting alpha-melanocyte-stimulating hormone in mice improved skin healing and reduced scarring.

Alopecia areata may be linked to hearing problems, so patients should monitor their hearing.

A specific group of early-stage melanocytes is reduced in vitiligo-affected skin, which may explain treatment resistance.

The paper concludes that understanding melanocyte development can help in insights into skin diseases and melanoma diversity.

Notch/RBP-J signaling is crucial for proper placement and timing of melanocyte development in hair follicles.

Melanocyte development from neural crest cells is complex and influenced by many factors, and better understanding could help treat skin disorders.

Melanocyte precursors in human fetal skin follow a specific migration pattern and some remain in the skin's deeper layers.

Hair graying may be caused by stem cell depletion from stress or melanocyte damage.

Repigmentation in vitiligo may come from melanocyte stem cells in the skin.

The study found increased skin pigmentation and variable melanocyte density in a patient with Addison's disease.

New treatments for vitiligo may focus on protecting melanocyte stem cells from stress and targeting specific pathways involved in the condition.

Repigmentation patterns in vitiligo depend on melanocyte source, lesion status, and therapy choice.

As people age, their hair follicles produce less pigment, leading to gray and white hair, due to factors like reduced enzyme activity and damage to melanocyte DNA.

Transplantation is effective for stable leucoderma but not for progressive, widespread vitiligo vulgaris.

Hair follicle stem cells are key for hair and skin regeneration, can be reprogrammed, and have potential therapeutic uses, but also carry a risk of cancer.

Stem cells in the hair follicle are regulated by their surrounding environment, which is important for hair growth.

We need more research on human hair follicle pigmentation, not just mouse models.

Melanocytes are crucial for skin pigmentation and can affect conditions like melanoma, vitiligo, and albinism, as well as hair color and hearing.

Human hair follicle stem cells can be isolated using specific markers for potential therapeutic use.

The conclusion is that tissue structure is key for stem cell communication and maintaining healthy tissues.