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Research 151–180 of 1000+
- The pathogenesis of alopecia areata
- Immunopathogenesis of Primary Cicatricial Alopecia
- Abstracts from the Symposium on the Pathophysiology of Hair Growth Organized by the Institute of Dermatology at the Royal Society of Medicine, 29 October 1987
- Molecular mechanisms of androgenetic alopecia
- The Effect of Plasma Rich in Growth Factors on Pattern Hair Loss: A Pilot Study
- Cicatricial (Scarring) Alopecias
- Molecular functional analyses revealed essential roles of HSP90 and lamin A/C in growth, migration, and self-aggregation of dermal papilla cells
- Anagen hair follicles transplanted into mature human scars remodel fibrotic tissue
- Function of KLF4 in Stem Cell Biology
- Research on the Mechanism of Hair Dyeing Agents and Strategies for Health Risks Reduction
- Low Level Laser and Light Therapy
- Sensitization to saw palmetto and minoxidil in separate topical extemporaneous treatments for androgenetic alopecia
- HHV-6A and HHV-6B in Drug-Induced Hypersensitivity Syndrome/Drug Reaction with Eosinophilia and Systemic Symptoms
- Minoxidil
- Alopecia Areata
- Lignans and Neolignans: Plant secondary metabolites as a reservoir of biologically active substances
- The Changing Landscape of Alopecia Areata: The Therapeutic Paradigm
- Role of β-Catenin Activation Levels and Fluctuations in Controlling Cell Fate
- Zinc: A complementary factor in the treatment of chronic hepatitis C? (Review)
- Female Adolescent Hair Disorders
- Comparison of the Therapeutic Effect of Allogeneic and Xenogeneic Small Extracellular Vesicles in Soft Tissue Repair
- Retinoids for Chemoprophylaxis of Nonmelanoma Skin Cancer
- CUTANEOUS T-CELL LYMPHOMA
- Immunomodulators in the treatment of cutaneous lymphoma
- Cutaneous Reactions to Drugs
- Dermoscopic features of some scalp disorders
- Gene Therapy: Recent Advances and Applications in Dermatology
- Cutaneous Scarring: Basic Science, Current Treatments, and Future Directions
- Cutaneous signs in COVID ‐19 patients: A review
- Human Scalp Hair Follicles Are Both a Target and a Source of Prolactin, which Serves as an Autocrine and/or Paracrine Promoter of Apoptosis-Driven Hair Follicle Regression