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Different types of sun exposure damage skin cells and immune cells, with chronic exposure leading to more severe and lasting damage.

AP-2α and AP-2β proteins are essential for healthy adult skin and hair.

The study found that stem cells and neutrophils are important for regenerating hair follicle structures in mice.

The biofilm-dispersing wound gel helps wounds heal faster and prevents infection.

A mouse gene mutation increases the risk of skin cancer.

Jagged-1 in skin Tregs is crucial for timely wound healing by recruiting specific immune cells.

Immune cells are essential for skin regeneration using biomaterial scaffolds.

Non-immune dermal cells dominate, epidermal cells increase after day 9, and certain immune cells persist beyond inflammation in wound-induced hair follicle regeneration.

Aged individuals heal wounds less effectively due to specific immune cell issues.

The technique effectively shows how human skin and hair cells form into ball-like structures.

Fat cells help recruit healing cells and build skin structure during wound healing.

Fat cells in the skin help start healing and form important repair cells after injury.

Macrophages help regrow hair by activating stem cells using AKT/β-catenin and TNF.

Regulatory T cells are essential for normal and improved wound healing in mice.

Blocking TSLP reduces skin inflammation and cell overgrowth in psoriasis.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

Helminth protein helps wounds heal better by reducing scarring and promoting tissue growth.

A protein called EGFR protects hair follicle stem cells, and when it's disrupted, hair follicles can be damaged, but blocking certain pathways can restore hair growth.

Ovol2 is crucial for hair growth and skin healing by controlling cell movement and growth.

Targeting TGFβ can improve skin immunity in older people.

IL-3 worsens lupus nephritis and blocking it improves kidney health.

When skin blisters, healing the wound is more important than growing hair, and certain stem cells mainly fix the blisters without helping hair growth.

High DHA levels delay wound healing and worsen skin repair quality.

Certain immune cells worsen post-surgery gut paralysis by activating a specific immune response.

The new scaffold with two growth factors speeds up skin healing and reduces scarring.

NSG mice had the most mites, and genetic factors affect immune response and susceptibility.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

Lack of Cisd2 disrupts calcium balance in cells, leading to poorly functioning neutrophils.

Different immune cells like platelets, mast cells, neutrophils, macrophages, T cells, B cells, and innate lymphoid cells all play roles in skin wound healing, but more research is needed due to inconsistent results and the complex nature of the immune response.

A chemical called 5-Bromo-2′-deoxyuridine caused rapid hair loss in mice by killing certain skin cells through a specific cell death pathway.