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Research 61–90 of 316
- Lrig1 Expression Defines a Distinct Multipotent Stem Cell Population in Mammalian Epidermis
- HPV8 Field Cancerization in a Transgenic Mouse Model Is due to Lrig1+ Keratinocyte Stem Cell Expansion
- Lrig1-expression confers suppressive function to CD4+ cells and is essential for averting autoimmunity via the Smad2/3/Foxp3 axis
- LRIG1 is a positive prognostic marker in Merkel cell carcinoma and Merkel cell carcinoma expresses epithelial stem cell markers
- Lrig1 Expression in Human Sebaceous Gland Tumors
- 1378 Identification of two distinct stem cell clusters, Lrig1-derived and Wnt/CD44-dependent, in corneal epithelium
- 078 The transmembrane protein LRIG1 regulates receptor tyrosine kinases in skin development and homeostasis
- Faculty Opinions recommendation of Lrig1 expression defines a distinct multipotent stem cell population in mammalian epidermis.
- O04 HPV8 E6 leads to Lrig1+ keratinocyte stem cell expansion
- 1373 Specific in vivo ablation of Lrig1-positive follicular stem cells results in sebaceous gland loss in mice
- The Roles of the Lrig1- and Lgr6-Positive Stem Cells in the Skin and the Functions of the Molecular Markers That Define Them
- Cutaneous gain-of-function mutation of LRIG3 leads to alopecia by upregulation of ERBB, PI3K/AKT, NOTCH1 signaling pathways
- 575 Senescent atrophic epidermis retains Lrig1+ stem cells and loses Wnt signaling, a phenotype shared with CD44KO mice
- Stem cell dynamics, migration and plasticity during wound healing
- Epidermal stem cell diversity and quiescence
- Epithelial stem cells and implications for wound repair
- Compartmentalized Epidermal Activation of β-Catenin Differentially Affects Lineage Reprogramming and Underlies Tumor Heterogeneity
- Strategic Targets in Acne: The Comedone Switch in Question
- Hair eruption initiates and commensal skin microbiota aggravate adverse events of anti-EGFR therapy
- Distinct Fibroblast Lineages Give Rise to NG2+ Pericyte Populations in Mouse Skin Development and Repair
- Atypical Protein Kinase C Isoform, aPKCλ, Is Essential for Maintaining Hair Follicle Stem Cell Quiescence
- Keratin 79 identifies a novel population of migratory epithelial cells that initiates hair canal morphogenesis and regeneration
- Chemically induced skin tumors arise from long-lived stem cells of the upper hair follicle
- 449 Long-Term Role of Hair Follicle Stem Cells in Epidermal Wound Healing
- Impact of Stem Cells on Reparative Regeneration in Abdominal and Dorsal Skin in the Rat
- Fondation René Touraine
- Single-cell analysis of 2,3,7,8-tetrachlorodibenzo- <i>p</i> -dioxin (TCDD)-treated murine skin demonstrates that sebaceous gland differentiation precedes <i>Ahr</i> -dependent seboatrophy with elevated expression of infundibular <i>Blimp1</i>
- 574 Murine sebaceous gland homeostasis requires intact cutaneous innervation in a hair cycle-dependent context
- Faculty Opinions recommendation of Keratin 79 identifies a novel population of migratory epithelial cells that initiates hair canal morphogenesis and regeneration.
- A cell polarity protein, aPKCλ, is essential for maintaining hair follicle stem cell quiescence and hair follicle regeneration