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Pediatric androgenetic alopecia is linked to obesity, family history, hormonal imbalances, and requires personalized treatment including managing comorbidities.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

A genetic defect in vitamin D receptors causes severe rickets and hair loss in children, but some heal as they age.

Taking too much vitamin E caused a young man's blood clotting problems, which improved after he stopped the vitamin E and got treatment.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Using cetirizine on the skin and taking vitamin D can help increase hair growth in children with hair loss from ectodermal dysplasia.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

People with telogen effluvium often have much lower vitamin D levels than healthy individuals.

Western diet may cause male pattern baldness; low glycemic diet with magnesium could help.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

Rubbing calcipotriol on the skin works better than taking vitamin D pills for treating mild to moderate alopecia areata.

The document concludes that more research is needed to confirm if blood tests for things like iron and vitamin D are helpful for hair loss from Telogen effluvium.

Vitamin D made by the skin plays a role in immune defense and skin health, and more research is needed to understand its full effects.

Vitamin D and calcium are important for skin stem cell function and wound healing.

Updated treatments for female hair loss include minoxidil, antiandrogens, hair transplants, and light therapy.

Low-dose UVB light improves hair growth effects of certain stem cells by increasing reactive oxygen species.

Even after successful weight loss surgery, a patient can have health issues like fatigue due to nutrient deficiencies and hormone imbalances.

The R343H mutation in the VDR gene causes vitamin D-resistant rickets with alopecia by impairing specific gene activity.

Gastroparesis may cause hair loss due to micronutrient deficiencies, and multivitamins might help.

Topical vitamin D is useful for some skin conditions but not effective for others, and more research is needed.

OCT binds strongly to hair sheath cells and may affect skin and hair growth with fewer side effects than vitamin D3.

A nutrition program improved a young woman's concussion symptoms.

Gene therapy with a vitamin D receptor gene improved hair growth in rats with a type of rickets-related baldness.

Alopecia areata incognito in children can be effectively treated with triamcinolone acetonide and supplements, leading to full hair regrowth.

VDR regulation varies by tissue and is crucial for its biological functions.

Men with early hair loss have lower vitamin D levels.

Redheaded people may have evolved to efficiently make vitamin D in areas with less sunlight.

The vitamin D receptor can act without its usual activating molecule, affecting hair growth and skin cancer, but its full range of actions is not well understood.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Glucocorticoids for progressive muscular dystrophy in children don't improve muscle strength or function but do increase certain side effects.